Author

B. Wiegmann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1

Frequent Co-Authors

S. Haid· 2 shared T. Pietschmann· 2 shared A. Haverich· 1 shared A. Krawczyk· 1 shared A. Matthaei· 1 shared A. P. Gunesch· 1 shared A.-M. Hitz· 1 shared B. Sitek· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersMicrobiology

S. Haid

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Donor tissue-resident memory-like T and NK cells generate a transient peripheral chimerism in lung transplant recipients, potentially protective from chronic lung allograft dysfunction

R. Bellmas-Sanz, A.-M. Hitz, B. Wiegmann, J. F. Kuehne, E. Chichelnitskiy, T. S. Kapellos, K.-A. Blaesing, J. Keil, K. Beushausen, W. Sommer, K. Haendler, M. Becker, K. Bassler, L.-M. Horn, D. Jonigk, , C. S. Falk

Donor TRM-like T and NK cells create a temporary chimerism in lung transplant recipients, possibly reducing the risk of chronic lung allograft dysfunction.

bioRxiv · Jun 16, 2023 · PreprintImmunology

Repurposing screen highlights broad-spectrum coronavirus antivirals and their host targets

S. Haid, A. Matthaei, M. Winkler, S. M. Sake, A. P. Gunesch, J. Rueckert, G. Vieyres, D. Kuehl, T.-T. Nguyen, L. Lasswitz, F. Zapatero, G. Brogden, G. Gerold, B. Wiegmann, U. Bilitewski, , T. Pietschmann

The paper screens a drug library to identify broad-spectrum coronavirus antivirals and their host targets, finding compounds that inhibit both alpha- and beta-coronaviruses.

bioRxiv · Jul 14, 2021 · PreprintMicrobiology

Differential interferon-α subtype immune signatures suppress SARS-CoV-2 infection

J. Schuhenn, T. L. Meister, D. Todt, T. Bracht, K. Schork, J.-N. Billaud, C. Elsner, N. Heinen, Z. Karakoese, S. Haid, S. Kumar, L. Brunotte, M. Eisenacher, J. Chen, Z. Yuan, , S. Pfaender

The paper identifies differential antiviral effects of IFN-alpha subtypes against SARS-CoV-2 and highlights IFN5's superior activity, suggesting therapeutic potential.

bioRxiv · May 20, 2021 · PreprintCell Biology