Author

D. Lemos

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. C. Figueroa-Ramirez· 1 shared A. G. Pletnev· 1 shared A. L. Ramirez· 1 shared A. Rampazzo· 1 shared A. Singapuri· 1 shared B. Paylor· 1 shared C. Chang· 1 shared C. E. Barkauskas· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperCell Biology

A. C. Figueroa-Ramirez

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3 papers

Recent·Most cited

Two sides of a coin: a Zika virus mutation selected in pregnant rhesus macaques promotes fetal infection in mice but at a cost of reduced fitness in nonpregnant macaques and diminished transmissibility by vectors

D. Lemos, J. B. Stuart, W. Louie, A. Singapuri, A. L. Ramirez, J. Watanabe, J. Usachenko, R. Keesler, C. Sanchez-San Martin, T. Li, C. Martyn, G. Oliveira, S. Saraf, N. D. Grubaugh, K. Andersen, , L. L. Coffey

This paper identifies a ZIKV mutation, M1404I, that enhances fetal infection in mice but reduces viral fitness and transmissibility in nonpregnant macaques and mosquitoes.

bioRxiv · Aug 11, 2020Microbiology

Orphan nuclear receptor COUP-TFII drives the myofibroblast metabolic shift leading to fibrosis

L. Li, P. Galichon, X. Xiao, A. C. Figueroa-Ramirez, D. Tamayo, J. J. Lee, M. Kalocsay, D. Gonzalez-Sanchez, M. S. Chancay, K. McCracken, D. Lemos, N. Lee, T. Ichimura, Y. Mori, M. T. Valerius, , J. V. Bonventre

This study identifies COUP-TFII as a driver of myofibroblast metabolic shift to aerobic glycolysis, promoting fibrosis in multiple organs.

bioRxiv · Jun 5, 2020 · PreprintCell Biology

Pathogenic potential of Hic1 expressing cardiac stromal progenitors

H. Soliman, B. Paylor, W. Scott, D. Lemos, C. Chang, M. Arostegui, M. Low, C. Lee, D. Fiore, P. Braghetta, V. Pospichalova, C. E. Barkauskas, V. Korinek, A. Rampazzo, K. MacLeod, , F. Rossi

This study identifies cardiac stromal progenitors as multipotent cells that contribute to fibrosis and arrhythmogenic cardiomyopathy under different pathological conditions.

bioRxiv · Feb 12, 2019Cell Biology