Author

E. Seto

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:proteomics· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

A. Chen· 1 shared A. Riva· 1 shared B. Fang· 1 shared C. Peng· 1 shared C. Zhang· 1 shared D. Dupere-Richer· 1 shared H. Lin· 1 shared H. Ling· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperCancer Biology

A. Chen

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

HDAC10 blockade upregulates SPARC expression thereby repressing melanoma cell growth and BRAF inhibitor resistance

H. Ling, Y. Li, C. Peng, S. Yang, E. Seto

This study shows that inhibiting HDAC10 increases SPARC expression, which suppresses melanoma growth and restores sensitivity to BRAF inhibitors.

bioRxiv · Dec 5, 2023 · PreprintCancer Biology

HDAC8-mediated inhibition of EP300 drives a neural crest-like transcriptional state that increases melanoma brain metastasis

M. F. Emmons, R. L. Bennett, A. Riva, C. Zhang, R. Macaulay, D. Dupere-Richer, B. Fang, E. Seto, J. Koomen, J. Li, A. Chen, P. Forsyth, J. Licht, K. S. M. Smalley

bioRxiv · Oct 12, 2022 · PreprintCancer Biology

HDAC11 regulates type I interferon signaling through defatty-acylation of SHMT2

J. Cao, L. Sun, P. Aramsangtienchai, N. A. Spiegelman, X. Zhang, E. Seto, H. Lin

This study identifies HDAC11 as a defatty-acylase that regulates type I interferon signaling by modifying SHMT2, revealing a novel function of HDAC11 beyond its deacetylase activity.

bioRxiv · Oct 31, 2017Biochemistry