Author

F. Abascal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Assay:whole-genome sequencing· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1

Frequent Co-Authors

F. MAURA· 2 shared O. Landgren· 2 shared V. Yellapantula· 2 shared Y. Zhang· 2 shared A. Damaskou· 1 shared A. Derkach· 1 shared A. Dogan· 1 shared A. Mikulasova· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

F. MAURA

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Molecular evolution of classic Hodgkin lymphoma revealed through whole genome sequencing of Hodgkin and Reed Sternberg cells

F. MAURA, B. Ziccheddu, J. J. Xiang, B. Bhinder, F. Abascal, K. Maclachlan, K. W. Eng, M. Uppal, F. He, W. Zhang, Q. Gao, V. Yellapantula, S. Park, M. Oberley, E. Ruckdeschel, , L. Giulino-Roth

This study uses whole genome sequencing of Hodgkin and Reed Sternberg cells to reconstruct the temporal evolution of classic Hodgkin lymphoma, identifying key mutations and structural variants.

bioRxiv · Nov 5, 2021Cancer Biology

The longitudinal dynamics and natural history of clonal haematopoiesis

M. A. Fabre, J. G. de Almeida, E. Fiorillo, E. Mitchell, A. Damaskou, J. Rak, V. Orru, M. Marongiu, M. Vijayabaskar, J. Baxter, C. Hardy, F. Abascal, M. S. Chapman, N. Williams, J. Nangalia, , G. S. Vassiliou

This study tracks clonal haematopoiesis clones over time, finding that mutation type influences growth rates and malignant risk.

bioRxiv · Aug 12, 2021Cancer Biology

Whole genome sequencing provides evidence of two biologically and clinically distinct entities of asymptomatic monoclonal gammopathies: progressive versus stable myeloma precursor condition

B. Oben, G. Froyen, K. H. Maclachlan, D. Leongamornlert, F. Abascal, B. Zheng-Lin, V. Yellapantula, A. Derkach, E. Geerdens, B. T. Diamond, I. Arijs, B. Maes, K. Vanhees, M. Hultcrantz, E. E. Manasanch, , F. Maura

The paper uses whole genome sequencing to identify two distinct myeloma precursor conditions, progressive and stable, based on genomic features.

bioRxiv · Nov 6, 2020 · PreprintCancer Biology