Author

G. Healey

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:CRISPR screen· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Strasser· 2 shared A. J. Kueh· 1 shared A. Janic· 1 shared A. Jeyasekharan· 1 shared A. T. Papenfuss· 1 shared A. Wei· 1 shared C. Koenig· 1 shared D. Kaloni· 1 shared

Activity by Year

2022-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCancer Biology

A. Strasser

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Author profile →

2 papers

Recent·Most cited

Combined absence of TRP53 target genes ZMAT3, PUMA and p21 cause a high incidence of cancer in mice

A. Janic, A. Strasser, M. Herold, M. S. Brennan, G. Healey, L. Gangoda, K. Brinkmann

The paper shows that combined absence of TRP53 target genes ZMAT3, PUMA, and p21 in mice leads to a higher incidence of cancer, with different tumor types depending on which genes are missing.

Cell Death & Differentiation · Dec 18, 2023Peer-reviewedCancer Biology

Genome-wide in vivo CRISPR screens identify GATOR1 as a potent tumor suppressor in MYC-driven lymphomagenesis

M. A. Potts, S. Mizutani, Y. Deng, S. Vaidyanathan, K. Ting, G. Giner, S. Shruti, Y. Liao, S. Diepstraten, A. J. Kueh, M. Pal, G. Healey, L. Tai, Z. Wang, C. Koenig, , M. J. Herold

The paper identifies GATOR1 as a tumor suppressor in MYC-driven lymphomagenesis using in vivo CRISPR screens and shows its sensitivity to mTOR inhibitors.

bioRxiv · Feb 16, 2022 · PreprintCancer Biology