J. Rismondo

This study identifies three adaptation mechanisms in Listeria monocytogenes to resist quaternary ammonium compounds BAC and CTAB through efflux pump overproduction, compensatory efflux systems, and membrane composition changes.

This study examines the growth of L. monocytogenes under salt and pH stress and identifies carbon sources and genes required for growth in Listeria synthetic medium.

This study shows that EslB deficiency in Listeria monocytogenes leads to thinner peptidoglycan and increased negative surface charge, enhancing lysozyme sensitivity.

This study identifies YngB as a UGPase in B. subtilis that contributes to WTA glucosylation and glycolipid formation under anaerobic conditions.

This study identifies the YtrBCDEF ABC transporter as a regulator of genetic competence and biofilm formation in Bacillus subtilis by affecting cell wall properties and ComK activity.

The paper identifies ReoM as a PASTA-eSTK substrate in Listeria monocytogenes, showing its phosphorylation is essential for controlling MurA degradation via ClpCP protease.

This study shows that EslB, a component of the ABC transporter EslABC, is essential for cell wall biosynthesis and modification in L. monocytogenes, affecting lysozyme resistance, cell division, and growth in high-sugar media.

This study demonstrates that WTA and LTA are enantiomeric polymers differentially cleaved by GlpQ, revealing stereochemical differences in their glycerol phosphate moieties.

This paper shows that GtcA is essential for LTA glycosylation in L. monocytogenes and B. subtilis and suggests it functions as a C55-P-sugar flippase.

This study identifies multiple FtsW and RodA enzymes in Listeria monocytogenes that maintain cell shape and antibiotic resistance, with their expression regulated in response to beta-lactam antibiotics.

This study identifies GpsB as a cytosolic adaptor that coordinates cell wall synthesis with the bacterial cell cycle by interacting with multiple enzymes and proteins.