Author

K. Ryman

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4

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Research Focus:host-pathogen & immune response· 4 Organism:C. elegans· 3 Approach:experimental assay· 2 Approach:genetic perturbation / CRISPR· 2 Organism:Drosophila· 1 Assay:imaging· 1 Assay:metabolomics· 1 Organism:mouse· 1

Frequent Co-Authors

P. O. Ljungdahl· 4 shared F. G. S. Silao· 3 shared M. Ward· 3 shared C. Chen· 2 shared N.-N. Liu· 2 shared T. Jiang· 2 shared A. Besold· 1 shared A. Broggi· 1 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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4 shared papersMicrobiology

P. O. Ljungdahl

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4 papers

Recent·Most cited

Proline catabolism is key to facilitating Candida albicans pathogenicity

F. G. S. Silao, T. Jiang, B. Bereczky-Veress, A. Kuehbacher, K. Ryman, N. Uwamohoro, S. Jenull, F. Nogueira, M. Ward, T. Lion, C. F. Urban, S. Rupp, K. Kuchler, C. Chen, C. Peuckert, P. O. Ljungdahl

This study shows that proline catabolism is essential for Candida albicans pathogenicity, as mutations in proline utilization genes reduce virulence in animal models.

bioRxiv · Jan 17, 2023 · PreprintMicrobiology

Glutamate dehydrogenase (Gdh2)-dependent alkalization is dispensable for escape from macrophages and virulence of Candida albicans

F. G. S. Silao, K. Ryman, T. Jiang, M. Ward, N. Hansmann, N.-N. Liu, C. Chen, P. O. Ljungdahl

This study shows that Gdh2-dependent alkalization is not required for C. albicans to escape macrophages or maintain virulence, challenging the existing model of amino acid catabolism in phagosomes.

bioRxiv · Jan 20, 2020Microbiology

Mitochondrial proline catabolism activates Ras1/cAMP/PKA-induced filamentation in Candida albicans

F. G. S. Silao, M. Ward, K. Ryman, A. Wallstrom, B. Brindefalk, K. Udekwu, P. O. Ljungdahl

This study shows that mitochondrial proline metabolism via PUT1 and PUT2 enhances ATP production, activates the Ras1/cAMP/PKA pathway, and promotes filamentation in C. albicans, contributing to virulence.

bioRxiv · Jul 23, 2018Microbiology

Intersection of phosphate transport, oxidative stress and TOR signalling in Candida albicans virulence

N.-N. Liu, P. Uppuluri, A. Broggi, A. Besold, K. Ryman, H. Kambara, N. Solis, V. Lorenz, W. Qi, M. Acosta Zaldivar, S. N. Emami, B. Bao, D. An, F. Bonilla, M. Sola-Visner, , J. R. Koehler

This study shows that Pho84 in Candida albicans affects virulence by modulating oxidative stress through Sod3 and TORC1 pathways.

bioRxiv · May 11, 2018Microbiology