Author

L. Hall-Stoodley

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 3 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:human· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

A. Forero· 2 shared A. Kenney· 2 shared A. Zani· 2 shared J. Kawahara· 2 shared J. Li· 2 shared J. S. Yount· 2 shared M. KC· 2 shared M. Lu· 2 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersImmunology

A. Forero

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Interferon-induced transmembrane protein 3 (IFITM3) limits lethality of SARS-CoV-2 in mice

A. Zani, A. Kenney, J. Kawahara, A. C. Eddy, X.-L. Wang, M. KC, M. Lu, E. A. Hemann, J. Li, M. E. Peeples, L. Hall-Stoodley, A. Forero, C. Cai, J. Ma, J. S. Yount

This study shows that IFITM3 protects mice from severe SARS-CoV-2 infection by limiting viral spread and inflammation, as demonstrated through knockout mouse models.

bioRxiv · Dec 22, 2021 · PreprintImmunology

Caspase-4/11 exacerbates disease severity in SARS-CoV-2 infection by promoting inflammation and thrombosis

M. Eltobgy, A. Zani, A. Kenney, S. Estfanous, E. Kim, A. Badr, C. Carafice, K. Daily, O. Whitham, M. Pietrzak, A. Webb, J. Kawahara, A. Eddy, P. Denz, M. Lu, , A. Amer

This study shows that caspase-4/11 worsens SARS-CoV-2 infection by increasing inflammation and thrombosis, independent of GSDMD, suggesting it as a potential drug target for severe COVID-19.

bioRxiv · Sep 24, 2021Immunology

Mycobacterium abscessus biofilms have viscoelastic properties which may contribute to their recalcitrance in chronic pulmonary infections.

E. S. Gloag, D. J. Wozniak, P. Stoodley, L. Hall-Stoodley

This study examines the viscoelastic properties of Mycobacterium abscessus biofilms and finds they may contribute to their persistence in chronic pulmonary infections.

bioRxiv · Oct 20, 2020Microbiology

Opposing activities of IFITM proteins in SARS-CoV-2 infection

G. Shi, A. D. Kenney, E. Kudryashova, L. Zhang, L. Hall-Stoodley, R. Robinson, D. Kudryashov, A. A. S. Compton, J. S. Yount

This study identifies opposing roles of IFITM3 in restricting and enhancing SARS-CoV-2 infection through distinct mechanisms involving endosomal restriction and plasma membrane fusion.

bioRxiv · Aug 11, 2020Immunology