Author

M. Panis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 5

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 5 Approach:experimental assay· 4 Assay:bulk RNA-seq· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

B. tenOever· 3 shared B. E. Nilsson-Payant· 2 shared B. Lee· 2 shared B. R. tenOever· 2 shared D. Blanco-Melo· 2 shared I. Golynker· 2 shared A. C. Borczuk· 1 shared A. Kodra· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersMicrobiology

B. tenOever

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

SARS-CoV-2 infection results in lasting and systemic perturbations post recovery

J. J. Frere, R. A. Serafini, K. D. Pryce, M. Zazhytska, K. Oishi, I. Golynker, M. Panis, J. Zimering, S. Horiuchi, D. A. Hoagland, R. Moller, A. Ruiz, J. B. Overdevest, A. Kodra, P. D. Canoll, , B. tenOever

This study compares short- and long-term systemic responses to SARS-CoV-2 and IAV in golden hamsters, finding persistent inflammation in olfactory tissues post-recovery.

bioRxiv · Jan 18, 2022Microbiology

Nipah virus Bangladesh infection elicits organ-specific innate and inflammatory responses in the marmoset model

C. S. Stevens, J. Lowry, T. Juelich, C. Atkins, K. Johnson, J. K. Smith, M. Panis, T. Ikegame, B. tenOever, A. N. Freiberg, B. Lee

This study investigates NiV Bangladesh infection in marmosets, revealing organ-specific innate and inflammatory responses through RNA-seq analysis.

bioRxiv · Oct 11, 2021Microbiology

SARS-CoV-2 hijacks p38β/MAPK11 to promote viral protein translation

C. A. Higgins, B. E. Nilsson-Payant, A. Kurland, C. Ye, T. M. Yaron, J. L. Johnson, B. Bonaventure, P. Adhikary, I. Golynker, B. R. Rosenberg, O. Danziger, M. Panis, L. C. Cantley, L. Martinez-Sobrido, B. R. tenOever, J. R. Johnson

This study identifies p38β as a critical host factor for SARS-CoV-2 replication and shows that inhibiting it reduces viral replication and cytokine production.

bioRxiv · Aug 20, 2021 · PreprintMicrobiology

SARS-CoV-2 launches a unique transcriptional signature from in vitro, ex vivo, and in vivo systems

D. Blanco-Melo, B. Nilsson-Payant, W.-C. Liu, R. Moeller, M. Panis, D. Sachs, R. Albrecht, B. R. tenOever

This study characterizes the host transcriptional response to SARS-CoV-2 and other respiratory viruses in multiple models, finding a unique muted response lacking certain cytokine and chemokine induction.

bioRxiv · Mar 24, 2020Microbiology

An inability to maintain the ribonucleoprotein genomic structure is responsible for host detection of negative-sense RNA viruses

D. Blanco-Melo, B. E. Nilsson-Payant, S. Uhl, B. Escudero, S. Olschewski, P. Thibault, M. Panis, M. Rosenthal, C. Munoz-Fontela, B. Lee, B. tenOever

The paper shows that limiting nucleoprotein (NP) in negative-sense RNA viruses leads to defective genomes and triggers host interferon responses, making NP a key antiviral target.

bioRxiv · Mar 12, 2020 · PreprintMicrobiology