Author

M. Poidinger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:ChIP-seq· 1

Frequent Co-Authors

A. Singhal· 3 shared J. Lum· 3 shared A. K. Pandey· 1 shared A. Larbi· 1 shared A. Lee· 1 shared A. Tay· 1 shared B. Lee· 1 shared C. Antoine Dutertre· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersImmunology

A. Singhal

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3 papers

Recent·Most cited

Proliferative and quiescent human gastric cancer stem-like cells are associated with differential chemoresistance and patient mortality

K. S. Ang, H. K. Lee, M. Chevrier, M. Goh, J. Ling, V. Koh, X. Zhang, J. C. Tan, N. Lee, S. Erdal Irac, Z. M. Lim, J. Lum, A. Tay, C. Antoine Dutertre, C. Tang, , J. Chen

This study identifies two stem-like gastric cancer subpopulations with distinct chemoresistance and survival outcomes using scRNA-seq and organoid models.

bioRxiv · Oct 23, 2020 · PreprintImmunology

Host cholesterol dependent activation of VapC12 toxin enriches persister population during Mycobacterium tuberculosis infection

A. K. Pandey, S. Talwar, M. Pandey, C. Sharma, R. Kutum, J. Lum, D. Carbajo, M. Poidinger, R. Goel, D. Dash, A. Singhal

This study identifies cholesterol-dependent activation of the VapC12 toxin in Mycobacterium tuberculosis as a mechanism that increases persister formation during infection.

bioRxiv · Nov 26, 2019 · PreprintMicrobiology

Histone acetylome-wide association study of tuberculosis

R. C. H. del Rosario, J. Poschmann, P. Kumar, C. Y. Cheng, S. T. Ong, H. S. Hajan, D. Kumar, M. Marzuki, X. Lu, A. Lee, Y. Tang, C. B. E. Chee, C. Lim, B. Lee, J. Lum, , S. Prabhakar

This study conducts a histone acetylome-wide association study (HAWAS) of tuberculosis, identifying differentially acetylated loci and their association with immune phenotypes.

bioRxiv · May 21, 2019 · PreprintImmunology