Author

P. Andersson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Animal Behavior & Cognition· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:human· 3 Approach:experimental assay· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

P. Huang· 2 shared Z. Amoozgar· 2 shared A. S. Kumar· 1 shared C. Chuang· 1 shared D. Fukumura· 1 shared D. Jones· 1 shared E. Pereira· 1 shared G. Ferraro· 1 shared

Activity by Year

2022-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

P. Huang

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Age differences in brain functional connectivity underlying proactive interference in working memory.

P. Andersson, M. Schrooten, J. Persson

This study examines age-related differences in resting-state functional connectivity linked to proactive interference in working memory using cross-sectional and longitudinal data.

bioRxiv · Jun 18, 2024Animal Behavior & Cognition

Breast cancer progression and metastasis to lymph nodes reveals cancer cell plasticity and MHC class II-mediated immune regulation

P.-J. Lei, E. Pereira, P. Andersson, Z. Amoozgar, J. W. V. Wijnbergen, M. J. O'Melia, H. Zhou, S. Chatterjee, W. W. Ho, J. M. Posada, A. S. Kumar, S. Morita, C. Chuang, I. Ergin, D. Jones, , T. P. Padera

This study identifies MHC class II expression in metastatic breast cancer cells and shows its role in immune evasion and tumor progression through IFN-gamma and JAK/STAT signaling.

bioRxiv · Oct 30, 2022Cancer Biology

Combined blockade of VEGF, Angiopoietin-2, and PD1 reprograms glioblastoma endothelial cells into quasi-antigen-presenting cells

Z. Amoozgar, J. Ren, N. Wang, T. P. Uccello, P. Andersson, G. Ferraro, S. Rajan, P. Lei, S. Subudhi, K. Kawaguchi, R. E. Tay, I. L. Gomes Dos Santos, P. Huang, H.-J. Kim, D. Fukumura, R. Jain

This study shows that blocking VEGF, Ang2, and PD1 reprograms GBM endothelial cells into quasi-antigen-presenting cells, enhancing anti-tumor responses and controlling edema.

bioRxiv · Sep 3, 2022 · PreprintCancer Biology