R. J. Whitaker

This study shows that phage-mediated resolution of genetic conflict in Pseudomonas aeruginosa lysogens leads to evolutionary changes in CRISPR immunity and genome structure.

This study identifies 1302 prophages in 21 Neisseria species using virus prediction tools and finds that 20% are significantly targeted by CRISPR-Cas systems, suggesting interspecies phage movement.

This paper identifies Phaeobacter phage MD18, a novel phage infecting Phaeobacter inhibens, and finds that type IV pili serve as receptors for phage adsorption.

The paper models CRISPR-mediated host-virus interactions and shows empirical systems exhibit weighted-nested protection networks indicative of host suppression of virus diversification.

This study identifies a viral anti-CRISPR enzyme that degrades cyclic tetra-adenylate, neutralizing type III CRISPR immunity in bacteria and archaea.

This paper models how antibiotic treatment can control bacterial infections by inducing latent phage, showing that sub-inhibitory antibiotics can manage infections when bacteria carry latent viruses.

The paper introduces a cell-centric metric to quantify viral invasion fitness, showing that strategies like lysogeny can outperform lytic strategies under certain conditions.

This study identifies essential genes for Sulfolobus islandicus growth, challenges previous assumptions about essential functions, and highlights genes with unknown roles.