Author

V. Ridger

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

C. Souilhol· 3 shared P. Evans· 3 shared H. Roddie· 2 shared J. Serbanovic-Canic· 2 shared L. Canham· 2 shared M. Ariaans· 2 shared M. FRAGIADAKI· 2 shared S. Francis· 2 shared

Activity by Year

2018-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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3 shared papersCell Biology

C. Souilhol

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

JAG1-NOTCH4 MECHANOSENSING DRIVES ATHEROSCLEROSIS

C. Souilhol, X. Li, L. Canham, H. Roddie, D. Pirri, B. Tardajos Ayllon, E. Chambers, M. Dunning, M. Ariaans, J. Li, Y. Fang, M. FRAGIADAKI, V. Ridger, J. Serbanovic-Canic, S. De Val, , P. Evans

This study shows JAG1-NOTCH4 signaling promotes atherosclerosis by repressing endothelial repair in response to low oscillatory shear stress.

bioRxiv · May 15, 2020Pathology

β1 integrin is a sensor of blood flow direction

I. Xanthis, C. Souilhol, J. Serbanovic-Canic, H. Roddie, A. Kalli, M. Fragiadaki, R. Wong, D. Shah, J. Askari, L. Canham, N. Akhtar, S. Feng, V. Ridger, J. Waltho, E. Pinteaux, , P. Evans

This study identifies β1 integrin as a sensor for blood flow direction in endothelial cells, showing it is activated by unidirectional but not bidirectional shear stress.

bioRxiv · Jan 3, 2019Cell Biology

Neutrophil microvesicles drive atherosclerosis by delivering miR-155 to atheroprone endothelium.

I. Gomez, B. Ward, C. Souilhol, C. Recarti, M. Ariaans, J. Johnston, A. Burnett, M. Mahmoud, L. A. Luong, L. West, M. Long, S. Parry, R. Woods, C. Hulston, B. Benedikter, , V. Ridger

The paper shows neutrophil microvesicles deliver miR-155 to atheroprone endothelium, promoting vascular inflammation and atherosclerosis in mice.

bioRxiv · May 24, 2018Cell Biology