Author

A. C. Bohrer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 4 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

K. D. Mayer-Barber· 4 shared D. L. Barber· 3 shared E. Castro· 3 shared P. J. Baker· 3 shared A. D. Klion· 2 shared B. B. Andrade· 2 shared C. E. Tocheny· 2 shared F. Torres-Juarez· 2 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersImmunology

K. D. Mayer-Barber

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4 papers

Recent·Most cited

The inflammatory microenvironment of the lung at the time of infection governs innate control of SARS-CoV-2 replication

P. J. Baker, A. C. Bohrer, E. Castro, E. P. Amaral, M. Snow-Smith, F. Torres-Juarez, S. T. Gould, A. T. Queiroz, E. R. Fukutani, C. M. Jordan, J. S. Khillan, K. Cho, D. L. Barber, B. B. Andrade, R. F. Johnson, , K. D. Mayer-Barber

This study shows that pre-existing lung inflammation reduces SARS-CoV-2 replication by activating innate immune pathways, highlighting the role of the inflammatory microenvironment in controlling viral infection.

bioRxiv · Mar 27, 2024Immunology

A TNF-IL-1 circuit controls Yersinia within intestinal granulomas

R. Matsuda, D. Sorobetea, J. Zhang, S. T. Peterson, J. P. Grayczyk, B. Herrmann, W. Yost, R. O'Neill, A. C. Bohrer, M. Lanza, C.-A. Assenmacher, K. D. Mayer-Barber, S. Shin, I. E. Brodsky

This study identifies a monocyte-intrinsic TNF-IL-1 circuit that controls Yersinia infection within intestinal granulomas by signaling to non-hematopoietic cells.

bioRxiv · Apr 21, 2023 · PreprintImmunology

Rapid GPR183-mediated recruitment of eosinophils to the lung after Mycobacterium tuberculosis infection

A. C. Bohrer, E. Castro, C. E. Tocheny, M. Assmann, B. Schwarz, E. Bohrnsen, M. A. Makiya, F. Legrand, K. L. Hilligan, P. J. Baker, F. Torres-Juarez, Z. Hu, H. Ma, L. Wang, L. Niu, , K. D. Mayer-Barber

This study shows that eosinophils migrate to the lungs early after Mycobacterium tuberculosis infection in mice and macaques, mediated by GPR183 and Ch25h, independent of CCR3.

bioRxiv · Feb 18, 2022Immunology

Eosinophils are an integral component of the pulmonary granulocyte response in Tuberculosis and promote host resistance in mice

A. C. Bohrer, E. Castro, Z. Hu, A. T. L. Queiroz, C. E. Tocheny, M. Assmann, S. Sakai, C. Nelson, P. J. Baker, H. Ma, L. Wang, W. Zilu, E. du Bruyn, C. Riou, K. D. Kauffman, , K. D. Mayer-Barber

This study shows eosinophils are recruited to tuberculosis-infected lung tissue in mice and contribute to host resistance against Mtb infection.

bioRxiv · May 29, 2021Immunology