Author

L. L. Lin

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Research Focus:variant calling· 1

Frequent Co-Authors

A. A. Jazaeri· 1 shared A. Jhingran· 1 shared A. Klopp· 1 shared A. M. Futreal· 1 shared A. Mitra· 1 shared A. Y. Delgado Medrano· 1 shared B. Pederson· 1 shared B. V. Chapman· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

A. A. Jazaeri

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

SEL1L-HRD1 interaction is prerequisite for the formation of a functional HRD1 ERAD complex

L. L. Lin, X. Wei, H. H. Wang, B. Pederson, M. Torres, Y. Lu, Z. J. Li, X. Liu, H. Mao, H. Wang, Z. Zhao, S. E. Sun, L. Qi

This study shows that the SEL1L-HRD1 interaction is essential for HRD1 ERAD complex function, as its disruption leads to developmental defects and cerebellar ataxia in mice.

bioRxiv · Apr 13, 2023 · PreprintBiochemistry

Dynamic evolution of cervical cancer mutations during chemoradiation using novel sampling approach

B. V. Chapman, T. Karpinets, T. T. Sims, G. Biegert, X. Wu, A. Y. Delgado Medrano, P. J. Eifel, A. Jhingran, L. L. Lin, L. M. Ramondetta, A. M. Futreal, A. A. Jazaeri, M. Frumovitz, K. M. Schmeler, J. Yue, , L. Colbert

This study uses whole exome sequencing to track cervical cancer mutations during chemoradiation, finding that CDK4/CDK6/cyclin D1-related mutations persist or increase, suggesting radiation resistance.

bioRxiv · Nov 25, 2019 · PreprintCancer Biology