Author

B. Ason

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:rat· 1

Frequent Co-Authors

X. Luo· 2 shared A. Bredemeyer· 1 shared A. Fu· 1 shared A. L. Koenig· 1 shared B. Kopecky· 1 shared C. Hale· 1 shared C. Jones· 1 shared C. Kuppe· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersImmunology

X. Luo

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Author profile →

2 papers

Recent·Most cited

Targeting the Immune-Fibrosis Axis in Myocardial Infarction and Heart Failure

J. M. Amrute, X. Luo, V. Penna, A. Bredemeyer, T. Yamawaki, G. S. Heo, S. Shi, A. L. Koenig, S. Yang, F. F. Kadyrov, C. Jones, C. Kuppe, B. Kopecky, S. Hayat, P. Ma, , K. Lavine

The paper identifies a fibroblast trajectory associated with cardiac fibrosis and shows that macrophage-fibroblast crosstalk via IL-1β signaling drives fibrosis.

bioRxiv · Oct 17, 2022 · PreprintImmunology

The evolving systemic biomarker milieu in obese ZSF1 rat model of human cardiometabolic syndrome: Characterization of the model and cardioprotective effect of GDF15

M. Stolina, X. Luo, D. Dwyer, C.-Y. Han, R. Chen, Y. Zhang, Y. Xiong, Y. Chen, J. Yin, B. Ason, C. Hale, M. M. Veniant

This study characterizes an obese ZSF1 rat model of cardiometabolic syndrome and shows that GDF15 treatment reduces metabolic markers and improves cardiac function.

bioRxiv · Mar 20, 2020Physiology