Author

C. Raiborg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 4 Biophysics· 1

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Research Focus:cell signaling & trafficking· 5 Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Assay:electronic health records· 2 Organism:human· 2 Assay:proteomics· 2 Approach:statistical model· 2

Frequent Co-Authors

H. Stenmark· 5 shared A. Brech· 3 shared E. M. Wenzel· 3 shared K. O. Schink· 2 shared S. W. Schultz· 2 shared A. Bellanger· 1 shared A. Carlson· 1 shared A. H. Lystad· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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5 shared papersCell Biology

H. Stenmark

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Lysosome repair by ER-mediated cholesterol transfer

M. Radulovic, E. M. Wenzel, S. Gilani, L. K. K. Holland, A. H. Lystad, S. Phuyal, V. M. Olkkonen, A. Brech, M. Jäättelä, K. Maeda, C. Raiborg, H. Stenmark

The paper identifies an ER-mediated cholesterol transfer mechanism for lysosome repair independent of ESCRTs, involving PI4K2A and OSBP.

bioRxiv · Sep 26, 2022 · PreprintCell Biology

ATPase activity of DFCP1 controls selective autophagy

V. Nähse, C. Raiborg, K. W. Tan, S. Mørk, M. L. Torgersen, E. M. Wenzel, M. Nager, V. T. Salo, T. Johansen, E. Ikonen, K. O. Schink, H. Stenmark

This study identifies DFCP1 as an ATPase essential for omegasome constriction and selective autophagy by facilitating autophagosome release.

bioRxiv · Feb 24, 2022Cell Biology

A phosphoinositide and RAB switch controls early macropinocytosis

H. Spangenberg, M. Sneeggen, M. Mateo Tortola, C. Valenzuela-Montenegro, Y.-Y. Chang, H. Stenmark, C. Raiborg, K. O. Schink

This study identifies a phosphoinositide and RAB switch that regulates macropinosome fate between endolysosomal maturation and a secretory route.

bioRxiv · Nov 3, 2021Cell Biology

Protein Crowding Mediates Membrane Remodeling in Upstream ESCRT-induced Formation of Intraluminal Vesicles

S. Liese, E. M. Wenzel, I. Kjos, R. V. Rojas Molina, S. W. Schultz, A. Brech, H. Stenmark, C. Raiborg, A. Carlson

This study shows upstream ESCRTs mediate membrane deformation and ILV formation through crowding and altering bending rigidity, without requiring ATP.

Proceedings of the National Academy of Sciences · Nov 2, 2020Peer-reviewedBiophysics

Unrestrained ESCRT-III drives chromosome fragmentation and micronuclear catastrophe

M. Vietri, S. W. Schultz, A. Bellanger, C. M. Jones, C. Raiborg, E. Skarpen, C. R. J. Pedurupillay, E. Kip, R. Timmer, A. Jain, P. Collas, R. L. Knorr, S. N. Grellscheid, H. Kusumaatmaja, A. Brech, , C. Campsteijn

This study shows that unrestrained ESCRT-III activity at micronuclei causes chromosome fragmentation and DNA damage, while it facilitates repair in primary nuclei.

bioRxiv · Jan 10, 2019Cell Biology