Author

E. Donato

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

A. Trumpp· 2 shared E. Espinet· 2 shared A. E. R. Martinez· 1 shared A. Fischer· 1 shared A. Schneeweiss· 1 shared C. C. Yeung· 1 shared C. H. Scheel· 1 shared C. Klein· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCancer Biology

A. Trumpp

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Single-cell profiling uncovers a Muc4-expressing metaplastic gastric cell type sustained by Helicobacter pylori-specific inflammation

V. P. O'Brien, Y. Kang, M. K. Shenoy, G. Finak, W. C. Young, J. Dubrulle, L. Koch, A. E. R. Martinez, J. A. Snow, J. Williams, E. Donato, S. K. Batra, C. C. Yeung, S. Bullman, M. A. Koch, , N. R. Salama

The paper identifies a Muc4-expressing metaplastic gastric cell type in mice driven by H. pylori inflammation and shows its association with proliferation in human gastric cancer.

bioRxiv · Dec 20, 2022 · PreprintMicrobiology

HAPLN1 is a driver for peritoneal carcinomatosis in pancreatic cancer

L. Wiedmann, F. De Angelis Rigotti, N. Vaquero-Siguero, E. Donato, E. Espinet, I. Moll, E. Alsina-Sanchis, R. Mülfarth, M. Vacca, J. Gerwing, A. Trumpp, A. Fischer, J. Rodriguez-Vita

This study identifies HAPLN1 as a driver of peritoneal metastasis in pancreatic cancer by enhancing tumor cell plasticity and immunomodulation.

bioRxiv · May 30, 2022Cancer Biology

Maintenance of epithelial traits and resistance to mesenchymal reprogramming promote proliferation in metastatic breast cancer

L. Eichelberger, M. Saini, H. D. Moreno, C. Klein, J. M. Bartsch, M. Falcone, M. Reitberger, E. Espinet, V. Vogel, E. Graf, T. Schwarzmayr, T.-M. Strom, M. Lehmann, M. Koenigshoff, N. Pfarr, , C. H. Scheel

This study shows that high EPCAM expression in metastatic breast cancer cells supports epithelial traits and plasticity, promoting metastatic outgrowth, while loss of epithelial traits inhibits it.

bioRxiv · Mar 19, 2020 · PreprintCancer Biology