Author

M. Falcone

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Organism:bacteria (other)· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Ciabattini· 1 shared A. Kabanova· 1 shared A. Schneeweiss· 1 shared A. Trumpp· 1 shared C. De Santi· 1 shared C. Giordano· 1 shared C. H. Scheel· 1 shared C. Klein· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperMicrobiology

A. Ciabattini

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Anti-capsule human monoclonal antibodies protect against hypervirulent and pandrug-resistant Klebsiella pneumoniae

E. Roscioli, V. Zucconi Galli Fonseca, S. S. Bosch, I. Paciello, G. Maccari, G. Batani, S. Stazzoni, G. Cardinali, G. Tiseo, C. Giordano, Y. Shen, L. Capoccia, M. Ridelfi, M. Troisi, N. Manganaro, , R. Rappuoli

This study isolates human monoclonal antibodies against hypervirulent, pandrug-resistant Klebsiella pneumoniae and identifies capsule-specific antibodies as protective against bloodstream infection.

bioRxiv · Feb 14, 2024 · PreprintMicrobiology

Maintenance of epithelial traits and resistance to mesenchymal reprogramming promote proliferation in metastatic breast cancer

L. Eichelberger, M. Saini, H. D. Moreno, C. Klein, J. M. Bartsch, M. Falcone, M. Reitberger, E. Espinet, V. Vogel, E. Graf, T. Schwarzmayr, T.-M. Strom, M. Lehmann, M. Koenigshoff, N. Pfarr, , C. H. Scheel

This study shows that high EPCAM expression in metastatic breast cancer cells supports epithelial traits and plasticity, promoting metastatic outgrowth, while loss of epithelial traits inhibits it.

bioRxiv · Mar 19, 2020 · PreprintCancer Biology

A Patient-Derived Cellular Model for Huntington Disease Reveals Phenotypes at Clinically Relevant CAG Lengths

C. L. K. Hung, T. Maiuri, L. E. Bowie, R. Gotesman, S. Son, M. Falcone, J. V. Giordano, V. Mattis, T. Lau, V. Kwan, V. Wheeler, J. Schertzer, K. Singh, R. Truant

This paper develops a patient-derived cellular model for Huntington Disease that maintains TP53 function and exhibits HD phenotypes at clinically relevant CAG lengths.

bioRxiv · Apr 12, 2018Cell Biology