Author

E. Graf

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Research Focus:metabolism & bioenergetics· 1 Organism:mouse· 1

Frequent Co-Authors

T. Schwarzmayr· 2 shared A. Boettcher· 1 shared A. Feuchtinger· 1 shared A. Israel· 1 shared A. Kukat· 1 shared A. M. Cypess· 1 shared A. Schneeweiss· 1 shared A. Trifunovic· 1 shared

Activity by Year

2020-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

T. Schwarzmayr

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Identification and characterization of distinct murine brown adipocyte lineages

S. Ussar, R. Karlina, D. Lutter, V. Miok, D. S. Fischer, I. Altun, T. Schoettl, K. Schorpp, A. Israel, C. Cero, J. W. Johnson, I. Kapser-Fischer, A. Boettcher, S. Keipert, A. Feuchtinger, , A. M. Cypess

The paper identifies and characterizes distinct murine brown adipocyte lineages using single-cell RNA sequencing and functional analysis.

bioRxiv · Aug 24, 2020 · PreprintCell Biology

Adaptive mitochondrial regulation of the proteasome

T. Meul, K. Berschneider, S. Schmitt, C. H. Mayr, L. F. Mattner, H. B. Schiller, A. Yazgili, X. Wang, C. Lukas, C. Prehn, J. Adamski, E. Graf, T. Schwarzmayr, F. Perocchi, A. Kukat, , S. Meiners

This study shows mitochondrial dysfunction reduces 26S proteasome assembly and activity, which can be reversed by aspartate or pyruvate supplementation.

bioRxiv · Apr 7, 2020 · PreprintCell Biology

Maintenance of epithelial traits and resistance to mesenchymal reprogramming promote proliferation in metastatic breast cancer

L. Eichelberger, M. Saini, H. D. Moreno, C. Klein, J. M. Bartsch, M. Falcone, M. Reitberger, E. Espinet, V. Vogel, E. Graf, T. Schwarzmayr, T.-M. Strom, M. Lehmann, M. Koenigshoff, N. Pfarr, , C. H. Scheel

This study shows that high EPCAM expression in metastatic breast cancer cells supports epithelial traits and plasticity, promoting metastatic outgrowth, while loss of epithelial traits inhibits it.

bioRxiv · Mar 19, 2020 · PreprintCancer Biology