Author

G. Eberl

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1

Frequent Co-Authors

M. Berard· 2 shared A. Charton· 1 shared A. Gewirtz· 1 shared B. Chassaing· 1 shared B. Polomack· 1 shared D. Corral· 1 shared D. Hudrisier· 1 shared D. R. Withers· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

M. Berard

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

ILC3s select for RORγt+ Tregs and establish tolerance to intestinal microbiota

M. Lyu, H. Suzuki, L. Kang, F. F. Gaspal, W. Zhou, J. Goc, L. Zhou, J. Z. Zhou, W. Zhang, J. Live Cell Bank, Z. Shen, J. G. Fox, R. Sockolow, T. M. Laufer, Y. Fan, , G. F. Sonnenberg

This study identifies ILC3s as key regulators that promote RORγt+ Tregs and suppress Th17 cells to maintain intestinal tolerance to microbiota.

bioRxiv · Apr 25, 2022Immunology

Metabolic regulation of ILC2 differentiation into ILC1-like cells during Mycobacterium tuberculosis infection

D. Hudrisier, D. Corral, A. Charton, M. Z. Krauss, E. Blanquart, F. Levillain, E. Lefrancais, T. Sneperger, J.-P. Girard, G. Eberl, Y. Poquet, J.-C. Guery, R. J. Arguello, M. R. Hepworth, O. Neyrolles

This study shows that Mycobacterium tuberculosis infection drives immature lung ILC2 to differentiate into ILC1-like cells through type 1 cytokines and a glycolytic program controlled by HIF1.

bioRxiv · Jan 19, 2021Immunology

Innate immune recognition of a bacterial MAMP leads to conditional activation of pro- or anti-inflammatory responses

J. H. Park, S. Cornick, G. Nigro, G. Bertin, F. Dejardin, R. Smits, M. Berard, F. Langa Vives, I. Gomperts Boneca, A. Gewirtz, B. Chassaing, N. Barnich, P. Sansonetti, G. Eberl

This study shows that flagellin, a bacterial MAMP, can trigger either pro- or anti-inflammatory responses depending on the tissue's physiological state and context.

bioRxiv · Jul 28, 2019 · PreprintImmunology

Excess calorie intake early in life increases susceptibility to colitis in the adult

Z. Alnabhani, S. Dulauroy, E. Lecuyer, B. Polomack, M. Berard, G. Eberl

The paper shows that early-life excess calorie intake increases adult susceptibility to colitis through mechanisms involving intestinal permeability, inflammation, and microbial hydrogen sulfide production.

bioRxiv · Jun 26, 2019 · PreprintImmunology