J. S. Johnson

Laterally transferred macrophage mitochondria acts as a signaling source promoting cancer cell proliferation

The paper shows that dysfunctional macrophage mitochondria transferred to cancer cells promote proliferation via ROS-induced ERK signaling.

Ten-year projection of white-nose syndrome disease dynamics at the southern leading-edge of infection in North America

The paper models the spread of white-nose syndrome in Texas over 10 years, finding northern populations more affected than southern ones due to environmental factors.

Winter activity of boreal bats

This study investigates winter activity and hibernation site selection of boreal bats using acoustic monitoring, finding increased activity near rock outcrops and root cellars, and a link between temperature and bat activity.

A genome-wide CRISPR-Cas9 screen identifies CENPJ as a host regulator of altered microtubule organization during Plasmodium liver infection.

Roosting ecology of the southernmost bats, Myotis chiloensis and Histiotus magellanicus, in southern Tierra del Fuego, Chile

This study examines day-roosting habits and thermoregulation in two bat species in southern Tierra del Fuego, finding frequent torpor use and habitat preferences linked to temperature and tree size.

Population connectivity predicts vulnerability to white-nose syndrome in the Chilean myotis (Myotis chiloensis) - a genomics approach

The paper uses genomics to analyze population structure and connectivity of Chilean myotis, finding isolation-by-distance and low connectivity, which may reduce disease spread risk.

A comprehensive map of the dendritic cell transcriptional network engaged upon innate sensing of HIV

This study maps the transcriptional network in HIV-1 infected dendritic cells, identifying key regulators of interferon response and maturation.

Ferroptosis-like signaling facilitates a potent innate defense against Plasmodium infection

This study shows that ferroptosis-like signaling controls Plasmodium liver infection by reducing parasite survival and enhancing host cell death.