Author

N. Arang

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Microbiology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Approach:genetic perturbation / CRISPR· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Organism:C. elegans· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

A. Aderem· 2 shared A. Kaushansky· 2 shared A. N. Douglass· 2 shared H. S. Kain· 2 shared J. S. Johnson· 2 shared A. J. Lewis· 1 shared A. Kishore· 1 shared A. Vaid· 1 shared

Activity by Year

2018-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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Curved chords = mutual co-authorship

2 shared papersCell Biology

A. Aderem

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

A kinome-wide synthetic lethal CRISPR/Cas9 screen reveals that mTOR inhibition prevents adaptive resistance to CDK4/CDK6 blockade in HNSCC

Y. Goto, K. Koshizuka, T. Ando, H. Izumi, X. Wu, K. Ford, X. Feng, Z. Wang, N. Arang, M. M. Allevato, A. Kishore, P. Mali, J. S. Gutkind

This study identifies mTOR inhibition as a way to prevent adaptive resistance to CDK4/6 blockade in HNSCC by targeting cell cycle pathways.

bioRxiv · Aug 7, 2023Cancer Biology

Intermittent hypoxia therapy engages multiple longevity pathways to double lifespan in C.elegans

J. N. Pitt, E. Chavez, K. M. Provencher, M. Chen, C. Tran, J. Tran, K. Huang, A. Vaid, M. L. Abadir, N. Arang, S. F. Leiser, M. B. Roth, M. Kaeberlein

This study shows intermittent hypoxia therapy doubles C. elegans lifespan without HIF-1 but requires DAF-16/FOXO and other phosphatidylinositol signaling components.

bioRxiv · Oct 13, 2022 · PreprintPhysiology

A genome-wide CRISPR-Cas9 screen identifies CENPJ as a host regulator of altered microtubule organization during Plasmodium liver infection.

K. Vijayan, N. Arang, L. Wei, R. Morrison, R. Geiger, R. K. Parks, A. J. Lewis, F. D. Mast, A. N. Douglass, H. S. Kain, J. D. Aitchison, J. S. Johnson, A. Aderem, A. Kaushansky

bioRxiv · Aug 31, 2020Cell Biology

Ferroptosis-like signaling facilitates a potent innate defense against Plasmodium infection

H. S. Kain, N. Arang, E. Glennon, A. N. Douglass, D. R. Dudgeon, J. S. Johnson, A. Aderem, A. Kaushansky

This study shows that ferroptosis-like signaling controls Plasmodium liver infection by reducing parasite survival and enhancing host cell death.

bioRxiv · Jan 31, 2018Microbiology