Author

K. Dixit

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 2 Immunology· 2 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Organism:human· 3 Research Focus:host-pathogen & immune response· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:clinical cohort / trial· 1

Frequent Co-Authors

H. Kumar· 2 shared K. Kim· 2 shared A. Chandele· 1 shared A. Patel· 1 shared A. R. Pico· 1 shared A. S. Neish· 1 shared A. Sharma· 1 shared A. Vishwakarma· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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2 shared papersBioengineering

H. Kumar

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Direct Ink Writing of Graphene Oxide Reinforced 13-93B1 Bioactive Glass Scaffolds for Bone Tissue Engineering Applications

K. Dixit, A. Vishwakarma, H. Kumar, K. Kim, N. Sinha

This paper fabricates graphene oxide-reinforced 13-93B1 bioactive glass scaffolds using direct ink writing and evaluates their mechanical, structural, and biological properties for bone tissue engineering.

bioRxiv · Mar 14, 2024 · PreprintBioengineering

Vascularized Liver Tissue Embedded Bioprinting Utilizing GelMA/Nanoclay-based Composite hydrogels

N. Tabatabaei Rezaei, K. Dixit, H. Kumar, J. John, G. Natale, S. S. Park, K. Kim

This study develops a GelMA/nanoclay composite hydrogel for vascularized liver tissue bioprinting, optimizing its properties to support cell behavior and perfusion.

bioRxiv · Nov 16, 2023 · PreprintBioengineering

A novel bivalent interaction mode underlies a non-catalytic mechanism for Pin1-mediated Protein Kinase C regulation

X.-R. Chen, K. Dixit, Y. Yang, M. I. McDermott, H. T. Imam, V. A. Bankaitis, T. I. Igumenova

This paper shows Pin1 regulates PKC through a non-catalytic mechanism by binding its C-terminal tail via a novel bivalent interaction mode.

bioRxiv · Sep 18, 2023Biophysics

Molecular basis of SARS-CoV-2 Omicron variant evasion from shared neutralizing antibody response

A. Patel, S. Kumar, L. Lai, C. Chakravarthy, R. Valanparambil, E. S. Reddy, K. Gottimukkala, P. Bajpai, D. R. Raju, V. V. Edara, M. E. Davis-Gardner, S. Linderman, K. Dixit, P. Sharma, G. Mantus, , E. A. Ortlund

The paper identifies three monoclonal antibodies that poorly neutralize the Omicron BA.1 variant, revealing structural and molecular features of antibody evasion.

bioRxiv · Oct 24, 2022Immunology

SARS-CoV-2 spike protein induces abnormal inflammatory blood clots neutralized by fibrin immunotherapy

J. K. Ryu, E. G. Sozmen, K. Dixit, M. Montano, Y. Matsui, Y. Liu, E. Helmy, T. J. Deerinck, Z. Yan, R. Schuck, R. M. Acevedo, C. M. Spencer, R. Thomas, A. R. Pico, S. S. Zamvil, , K. Akassoglou

This study shows SARS-CoV-2 Spike protein causes abnormal blood clots and thromboinflammation, which are neutralized by a fibrin-targeting antibody.

bioRxiv · Oct 12, 2021 · PreprintImmunology