Author

M. C. Basil

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell-cell communication· 1 Organism:human· 1

Frequent Co-Authors

E. Cantu· 3 shared A. E. Vaughan· 2 shared E. E. Morrisey· 2 shared X. Li· 2 shared A. I. Weiner· 1 shared A. J. Paris· 1 shared A. Nottingham· 1 shared B. J. Banaschewski· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersCell Biology

E. Cantu

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Vascular Endothelial-derived SPARCL1 Exacerbates Viral Pneumonia Through Pro-Inflammatory Macrophage Activation

G. Zhao, M. E. Gentile, L. Xue, C. V. Cosgriff, A. I. Weiner, S. Adams-Tzivelekidis, J. Wong, X. Li, S. Kass-Gergi, N. P. Holcomb, M. C. Basil, K. M. Steward, J. D. Planer, E. Cantu, J. D. Christi, , A. E. Vaughan

This study identifies SPARCL1 as a pro-inflammatory mediator secreted by endothelial cells that worsens viral pneumonia by promoting macrophage activation and is elevated in severe COVID-19 cases.

Nature Communications · May 18, 2024Peer-reviewedImmunology

An injury-induced tissue niche shaped by mesenchymal plasticity coordinates the regenerative and disease response in the lung

D. L. Jones, M. P. Morley, X. Li, Y. Yun, F. L. Cardenas-Diaz, S. Li, S. Zhou, S. E. Schaefer, U. V. Chembazhi, A. Nottingham, S. Lin, E. Cantu, J. M. Diamond, M. C. Basil, A. E. Vaughan, E. E. Morrisey

The paper identifies an injury-induced tissue niche (iTCH) in the lung that coordinates regenerative and disease responses through mesenchymal plasticity and signaling pathways.

bioRxiv · Feb 26, 2024 · PreprintCell Biology

Dysregulated alveolar epithelial cell progenitor function and identity in Hermansky-Pudlak syndrome

J. Y. Wang, N. S. Michki, S. Sitaraman, B. J. Banaschewski, R. Jamal, J. J. Gokey, S. M. Lin, J. B. Katzen, M. C. Basil, E. Cantu, J. A. Kropski, J. A. Zepp, D. B. Frank, L. R. Young

This study identifies intrinsic AT2 progenitor dysfunction and p53 pathway dysregulation as novel mechanisms driving pulmonary fibrosis in Hermansky-Pudlak syndrome.

bioRxiv · Jun 17, 2023Cell Biology

Type II alveolar cells with constitutive expression of MHCII and limited antigen presentation capacity contribute to improved respiratory viral disease outcomes

S. A. Toulmin, C. Bhadiadra, A. J. Paris, J. H. Lin, J. Katzen, M. C. Basil, E. E. Morrisey, G. S. Worthen, L. C. Eisenlohr

This study shows that Type II alveolar cells constitutively express MHCII but have limited antigen presentation capacity, which may help modulate immune responses without excessive inflammation during respiratory viral infections.

bioRxiv · Mar 18, 2021 · PreprintImmunology