Author

M. Corr

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Tao· 2 shared D. S. Herdman· 2 shared E. Raz· 2 shared J. Lee· 2 shared N. J. G. Webster· 2 shared A. J. A. Molina· 1 shared B. Nürnberg· 1 shared C. Benner· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

A. Tao

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Activation of the TRIF pathway and downstream targets results in the development of precancerous lesions during infection with Helicobacter.

P. Bali, I. Lozano-Pope, J. Hernandez, M. V. Estrada, M. Corr, M. A. Turner, M. Bouvet, C. Benner, M. Obonyo

This study shows that activation of the TRIF pathway and downstream IFN-I signaling promotes H. felis-induced gastric disease progression and is linked to poor survival in gastric cancer.

bioRxiv · Jun 4, 2023 · PreprintCancer Biology

Gnas ablation in CD11c+ cells prevents high-fat diet-induced obesity by elevating adipose tissue catecholamine levels and thermogenesis

L. Zeng, D. S. Herdman, J. Lee, A. Tao, M. Das, S. Bertin, L. Eckmann, S. K. Mahata, S. Devulapalli, H. Patel, A. J. A. Molina, O. Osborn, M. Corr, E. Raz, N. J. G. Webster

bioRxiv · Jan 27, 2022 · PreprintImmunology

Inhibition of IRF4 in dendritic cells by PRR-independent and -dependent signals inhibit Th2 and promote Th17 responses

J. Lee, J. Zhang, Y.-J. Chung, J. H. Kim, C. M. Kook, J. M. Gonzalez-Navajas, D. S. Herdman, B. Nürnberg, P. Insel, M. Corr, A. Tao, K. Yasuda, I. R. Rifkin, D. H. Broide, R. Sciammas, , E. Raz

The paper shows that cAMP-PKA-CREB signaling in cDC2s represses IRF4 and KLF4, promoting Th17 responses and inhibiting Th2, with similar effects observed through PRR-dependent signals like curdlan.

bioRxiv · Jun 28, 2019Immunology