Author

M. Doshi

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:drug response / drug discovery· 1 Assay:electrophysiology· 1 Approach:statistical model· 1 Assay:whole-exome sequencing· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. J. Murphy· 1 shared A. L. Christie· 1 shared A. L. Hong· 1 shared B. A. Leeper· 1 shared B. Crompton· 1 shared B. D. Kynnap· 1 shared B. Deng· 1 shared B. Dwivedi· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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1 shared paperCancer Biology

A. J. Murphy

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Author profile →

3 papers

Recent·Most cited

Transparent and stretchable metal nanowire composite recording microelectrode arrays

Z. Chen, K. Nguyen, G. Kowalik, X. Shi, J. Tian, M. Doshi, B. R. Alber, X. Ning, M. W. Kay, L. Lu

This paper develops stretchable, transparent microelectrode arrays using gold-coated silver nanowires, demonstrating high optical transparency, low impedance, and stable performance for biomedical applications.

bioRxiv · Oct 11, 2022Bioengineering

Targeting TRIP13 in Wilms Tumor with Nuclear Export Inhibitors

K. Mittal, B. P. Lee, G. W. Cooper, J. Shim, H. C. Jonus, W. J. Kim, M. Doshi, D. Almanza, B. D. Kynnap, A. L. Christie, X. Yang, G. S. Cowley, B. A. Leeper, C. L. Morton, B. Dwivedi, , A. L. Hong

This study identifies XPO1 inhibition with selinexor as a potential therapy for favorable histology Wilms tumors, showing synergy with doxorubicin in preclinical models.

bioRxiv · Feb 23, 2022Cancer Biology

Replication gaps underlie BRCA-deficiency and therapy response

N. J. Panzarino, J. Krais, M. Peng, M. Mosqueda, S. Nayak, S. Bond, J. Calvo, K. Cong, M. Doshi, M. Bere, J. Ou, B. Deng, L. J. Zhu, N. Johnson, S. Cantor

This study challenges the existing model of BRCA-deficiency hypersensitivity to chemotherapy by proposing that ssDNA replication gaps, rather than DNA double strand breaks or stalled forks, underlie the therapeutic response.

bioRxiv · Sep 25, 2019Cancer Biology