Author

M. Kinter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:metabolism & bioenergetics· 2 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Batushansky· 1 shared A. Cardoso· 1 shared A. Elnwasany· 1 shared A. H. Pereira· 1 shared A. Lemoff· 1 shared A. Pranay· 1 shared A. Richardson· 1 shared A. Tran· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

A. Batushansky

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Non-Necroptotic Roles of MLKL in Diet-Induced Obesity, Liver Pathology, and Insulin Sensitivity: Insights from a High Fat, High Fructose, High Cholesterol Diet Mouse Model

P. Ohene-Marfo, H. V. M. Nguyen, S. Mohammed, N. Thadathil, A. Tran, E. H. Nicklas, D. Wang, R. Selvarani, J. Farriester, R. Varshney, M. Kinter, A. Richardson, M. Rudolph, S. S. Deepa

This study investigates MLKL's non-necroptotic roles in diet-induced obesity and liver disease using a HFHFrHC mouse model, finding dose-dependent effects on inflammation and metabolic parameters.

bioRxiv · Jan 10, 2024Biochemistry

Loss of cardiac PFKFB2 drives Metabolic, Functional, and Electrophysiological Remodeling in the Heart

K. M. Harold, S. Matsuzaki, A. Pranay, B. L. Loveland, A. Batushansky, M. F. Mendez Garcia, C. A. Eyster, S. Stavrakis, Y. A. Chiao, M. Kinter, K. M. Humphries

This study shows that loss of cardiac PFKFB2 leads to metabolic, functional, and electrophysiological changes in the heart, including impaired systolic function and altered electrophysiology.

bioRxiv · Nov 22, 2023Physiology

Angiocrine IGFBP3 Spatially Coordinates IGF Signaling During Neonatal Cardiac Regeneration

S. Ali, W. Elhelaly, N. U. N. Nguyen, S. Li, I. Menendez-Montes, Z. Wang, M. Cui, A. Elnwasany, F. Xiao, S. Thet, N. Lam, A. Cardoso, A. H. Pereira, M. Goodarzi, M. Kinter, , H. Sadek

The paper identifies IGFBP3 as a secreted protein that spatially coordinates IGF signaling to promote neonatal cardiac regeneration.

bioRxiv · Sep 16, 2021Cell Biology