M.-C. Groleau

The paper identifies PfsE as a Pf phage protein that inhibits PQS quorum sensing and type IV pili extension, enhancing viral replication and protecting the host from competing phages.

This paper shows that Pseudomonas aeruginosa produces 3-oxo-C12-HSL without LasR during surface growth, upregulating virulence factors in neighboring cells.

This study identifies O2 limitations as the main factor promoting the emergence of Small Colony Variants (SCVs) in Pseudomonas aeruginosa, which helps the bacterium survive by forming biofilms to access electron acceptors.

This study develops a polymicrobial biofilm model of the CF lung to investigate community-specific antimicrobial resistance and identifies LasR loss-of-function as a driver of tobramycin tolerance through increased phenazine production.

The paper shows that Pseudomonas aeruginosa provides rhamnolipids enabling Burkholderia cenocepacia to swarm, and aflagellated PA14 allows both to colonize surfaces together.

The study identifies frequent LasR-defective Pseudomonas aeruginosa isolates in diverse environments, suggesting a widespread adaptation mechanism.

This study shows that Pseudomonas aeruginosa strains from non-clinical environments can adopt a SCV-like phenotype under lab conditions, suggesting this ability is conserved across strains.

This study uses experimental evolution to identify mutations in gacA and rsmA that rescue a swarming defect in a Pseudomonas aeruginosa mutant, confirming RsmA's central role in social motility.

This study identifies N-oxidated methyl-alkenylquinolines (HMAQ-NOs) as antimicrobial compounds produced by Burkholderia thailandensis, showing they are 50-fold more active than their non-oxidized counterparts against Bacillus subtilis.

The paper shows that phenylacetyl-CoA, not phenylacetic acid, suppresses virulence in Burkholderia cenocepacia by indirectly affecting CepIR-regulated pathways.

This study identifies two rsaM homologues in Burkholderia thailandensis that regulate quorum sensing systems by repressing specific AHL biosynthesis pathways.