Author

S. Urfer

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:host-pathogen & immune response· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Organism:human· 1

Frequent Co-Authors

D. Macabrey· 2 shared F. Allagnat· 2 shared M. Lambelet· 2 shared S. Deglise· 2 shared A. Bitto· 1 shared A. Longchamp· 1 shared A. S. Grillo· 1 shared B. M. G. Nguyen· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersPhysiology

D. Macabrey

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The hydrogen sulfide donor sodium thiosulfate limits inflammation but aggravate smooth muscle cells apoptosis and aneurysm progression in a mouse model of abdominal aortic aneurysm

C. Bechelli, D. Macabrey, F. Caloz, S. Urfer, M. Lambelet, F. Allagnat, S. Deglise

This study shows that sodium thiosulfate (STS), a hydrogen sulfide donor, reduces inflammation but worsens abdominal aortic aneurysm (AAA) progression by promoting smooth muscle cell apoptosis and ECM breakdown in mice.

bioRxiv · Sep 15, 2023 · PreprintPhysiology

Acarbose suppresses symptoms of mitochondrial disease in a mouse model of Leigh Syndrome

A. Bitto, A. S. Grillo, I. B. Stanaway, B. M. G. Nguyen, K. Ying, H. Tung, K. Smith, N. Tran, G. Velikanje, S. Urfer, J. M. Snyder, E.-B. Kayser, L. Wang, D. L. Smith, J. W. Thompson, , M. Kaeberlein

This study shows acarbose reduces symptoms and improves survival in a mouse model of Leigh Syndrome, independent of mTOR inhibition, and suggests gut microbiome modulation may impact mitochondrial disease.

bioRxiv · Jan 31, 2022 · PreprintBiochemistry

Sodium Thiosulfate acts as an H2S mimetic to prevent intimal hyperplasia via inhibition of tubulin polymerization

D. Macabrey, A. Longchamp, M. Macarthur, M. Lambelet, S. Urfer, J.-M. Corpataux, S. Deglise, F. ALLAGNAT

This study shows sodium thiosulfate prevents intimal hyperplasia by acting as an H2S mimetic, inhibiting VSMC proliferation and migration through microtubule depolymerization.

bioRxiv · Sep 9, 2021 · PreprintPhysiology