Author

T. Guan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Kuhlmann· 1 shared A. Xu· 1 shared B. Miller· 1 shared C. Li· 1 shared C. Perry· 1 shared C. Robles-Oteiza· 1 shared D. Ayeni· 1 shared D. Pei· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. Kuhlmann

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Author profile →

4 papers

Recent·Most cited

Aberrant Lipid Accumulation and Retinal Pigmental Epithelium Dysfunction in PRCD-deficient mice

S. I. Motipally, D. R. Kolson, T. Guan, S. Kolandaivelu

This study investigates PRCD-deficient mice and finds increased retinal cholesterol and RPE dysfunction resembling AMD.

bioRxiv · Mar 8, 2024 · PreprintCell Biology

Epigenetic Reshaping through Damage: Promoting Cell Fate Transition by BrdU and IdU Incorporation

C. Li, S. Chen, X. Xu, T. Guan, A. Xu, C. Li, S. Yu, H. Wu, D. Pei, L. Jing

The paper investigates how BrdU and IdU incorporation through DNA damage repair induces epigenetic changes that promote cell fate transition.

bioRxiv · Aug 28, 2023Cell Biology

Mass Spectrometry based Metabolomics Deciphered Metabolic Reprogramming That Was Required for Biofilm formation in Uropathogenic Escherichia coli

H. Lu, Y. Que, X. Wu, T. Guan, H. Guo

The paper uses metabolomics to identify metabolic changes required for biofilm formation in Uropathogenic Escherichia coli.

bioRxiv · Jun 17, 2019 · PreprintMicrobiology

Tumor regression mediated by oncogene withdrawal or erlotinib stimulates infiltration ofinflammatory immune cells in EGFR mutant lung tumors

D. Ayeni, B. Miller, A. Kuhlmann, P.-C. Ho, C. Robles-Oteiza, M. Gaefele, S. Levy, F. de Miguel, C. Perry, T. Guan, D. Zelterman, R. Homer, Z. Liu, S. Kaech, K. Politi

The paper shows that erlotinib and oncogene withdrawal in EGFR mutant lung tumors increase infiltration of inflammatory immune cells, suggesting immunostimulatory effects of tumor regression.

bioRxiv · Jan 27, 2018Cancer Biology