Author
D. I. Godfrey
Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.
Biology Disciplines
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2018-2023 · peak 2/yrCo-Author Collaboration Network
10 top collaborators · 39 mutual co-author links6 papers
Recent·Most cited
- DOI 10.1016/j.omtm.2024.101380
- ImmunologybioRxiv
Vgamma9Vdelta2 T cells recognize butyrophilin 2A1 and 3A1 heteromers
T. S. Fulford, C. Soliman, R. G. Castle…·A. P. Uldrich
The paper reveals that Vgamma9Vdelta2 T cells recognize BTN2A1 and BTN3A1 heteromers through dual epitope engagement, uncovering a new mechanism of immune activation.
DOI 10.1038/s41590-024-01892-z - ImmunologybioRxivPreprint
Host Immunity to Mycobacterium tuberculosis Infection is Similar in Simian Immunodeficiency Virus (SIV)-infected, Antiretroviral Therapy-treated and SIV-naïve Juvenile Macaques
E. C. Larson, A. L. Ellis-Connell, M. Rodgers…·C. A. Scanga
This study compares host immunity to Mycobacterium tuberculosis in SIV-infected, ART-treated and SIV-naïve juvenile macaques, finding similar TB progression and T cell responses.
DOI 10.1101/2022.12.14.520525 - ImmunologybioRxiv
CD36 family members are TCR-independent ligands for CD1 antigen-presentingmolecules
N. A. Gherardin, S. J. Redmond, H. E. McWilliam…·D. I. Godfrey
The paper identifies CD36 family members as ligands for CD1 molecules and shows that blocking this interaction enables accurate identification of CD1-restricted T cells.
DOI 10.1126/sciimmunol.abg4176 - ImmunologybioRxiv
Benzofuran sulfonates and small self-lipid antigens activate type II NKT cells via CD1d
C. F. Almeida, D. Smith, T.-Y. Cheng…·D. I. Godfrey
This study identifies benzofuran sulfonates and small self-lipid antigens as activators of type II NKT cells via CD1d, revealing non-lipidic small molecules that trigger CD1d-restricted NKT cell responses.
DOI 10.1073/pnas.2104420118 - ImmunologybioRxiv
MAIT cells contribute to protection against lethal influenza infection in vivo
B. van Wilgenburg, L. Loh, Z. Chen…·T. Hinks
This study shows MAIT cells protect against lethal influenza infection by accumulating and being activated early, with their absence increasing mortality in mice.
DOI 10.1038/s41467-018-07207-9