Author

E. Collisson

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:chromatin & epigenetic regulation· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Approach:deep learning· 1

Frequent Co-Authors

A. B. Olshen· 1 shared A. Casabianca· 1 shared A. H. Shain· 1 shared A. Renslo· 1 shared A. Roberts· 1 shared A. Sharma· 1 shared A. Zaman· 1 shared B. C. Hann· 1 shared

Activity by Year

2019-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 14 mutual co-author links

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1 shared paperCancer Biology

A. B. Olshen

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Exploiting KRAS-driven Ferroaddiction in Cancer Through Ferrous Iron-Activatable Drug Conjugates (FeADC)

H. Jiang, R. K. Muir, R. L. Gonciarz, A. B. Olshen, I. Yeh, B. C. Hann, N. Zhao, Y.-h. Wang, J. Korkola, S. C. Behr, M. J. Evans, e. collisson, A. Renslo

This paper develops FeADCs, ferrous iron-activatable drug conjugates, to target KRAS-driven cancers with improved tumor selectivity and systemic tolerability.

bioRxiv · May 12, 2020Cancer Biology

SMARCA4 supports the oncogenic landscape of KRAS-driven lung tumors

E. Collisson

The paper shows that SMARCA4 supports oncogenic processes in KRAS-driven lung tumors and suggests targeting SWI/SNF complexes for therapy.

bioRxiv · Apr 18, 2020 · PreprintCancer Biology

A Novel Model of Pancreatic Cancer Dormancy Reveals Mechanistic Insights and a Dormancy Gene Signature with Human Relevance

C. Dudgeon, C. R. Harris, Y. Chen, B. Ghaddar, A. Sharma, M. M. Shah, A. Roberts, A. Casabianca, E. Collisson, V. Balachandran, P. Vertino, S. De, D. R. Carpizo

This study introduces a novel pancreatic cancer dormancy model, identifies a dormancy gene signature, and reveals mechanisms including chromatin remodeling and CCL5 as a novel marker.

bioRxiv · Apr 13, 2020 · PreprintCancer Biology

Global analysis of epigenetic heterogeneity identifies divergent drivers of esophageal squamous cell carcinoma

W. CAO, H. Lee, W. Wu, A. Zaman, S. McCorkle, M. Yan, J. Chen, Q. Xing, N. Sinnott-Armstrong, H. Xu, M. R. Sailani, W. Tang, Y. Cui, J. Liu, H. Guan, , T. G. Bivona

The paper identifies epigenetic drivers of esophageal squamous cell carcinoma through multi-omics analysis, revealing hypomethylation patterns and non-canonical WNT signaling activation.

bioRxiv · May 18, 2019 · PreprintCell Biology