Author

E. Forest

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Organism:Drosophila· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. A. Watson· 1 shared A. Arias-Rojas· 1 shared A. Arifah· 1 shared A. David· 1 shared A. Di Cicco· 1 shared A. Djiane· 1 shared A. Houdusse· 1 shared A. L. Chambers· 1 shared

Activity by Year

2017-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 8 mutual co-author links

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Curved chords = mutual co-authorship

1 shared paperBiochemistry

A. A. Watson

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

MprF-mediated immune evasion is necessary for Lactiplantibacillus plantarum resilience in Drosophila gut during inflammation

A. Arias-Rojas, A. Arifah, G. Angelidou, B. Alshaar, U. Schombel, E. Forest, D. Frahm, V. Brinkmann, N. Paczia, C. Beisel, N. Gisch, I. Iatsenko

This study shows that MprF in Lactiplantibacillus plantarum helps it survive in Drosophila gut during inflammation by reducing immune response and maintaining microbial stability.

bioRxiv · Jan 9, 2024 · PreprintMicrobiology

How Myosin VI Traps its Off-State, is Activated and Dimerizes

L. Canon, C. Kikuti, V. J. Planelles-Herrero, T. Lin, F. Mayeux, H. Sirkia, Y. i. Lee, L. Heidsieck, L. Velikovsky, A. David, X. Liu, D. Moussaoui, E. Forest, P. Hook, K. J. Petersen, , A. Houdusse

This study investigates how Myosin VI is trapped in its off-state, activated, and dimerizes through structural and functional analyses of its interactions with partners.

bioRxiv · Jun 30, 2023Biochemistry

MAGI1 inhibits the AMOTL2/p38 stress pathway and prevents luminal breast tumorigenesis

D. Kantar, E. Bousquet Mur, M. Mancini, V. Slaninova, Y. Ben Salah, L. Costa, E. Forest, P. Lassus, C. Geminard, F. Boissiere-Michot, B. Orsetti, C. Theillet, J. Colinge, C. Benistant, A. Maraver, , A. Djiane

The paper shows that MAGI1 loss in luminal breast cancer promotes tumorigenesis by activating the AMOTL2/p38 stress pathway, which is inhibited by MAGI1.

bioRxiv · Jun 13, 2020Cancer Biology

Architecture of TAF11/TAF13/TBP complex suggests novel regulatory state in General Transcription Factor TFIID function

K. Gupta, A. A. Watson, T. Baptista, E. Scheer, A. L. Chambers, C. Koehler, J. Zou, I. Obong-Ebong, E. Kandiah, A. Temblador, A. Round, E. Forest, P. Man, C. Bieniossek, E. D. Laue, , I. Berger

The paper identifies a ternary TAF11/TAF13/TBP complex and reveals a novel regulatory state in TFIID function through structural and biochemical analyses.

bioRxiv · Jul 26, 2017Biochemistry