Author

J. DasSarma

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biophysics· 1 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:imaging· 1

Frequent Co-Authors

A. Behera· 1 shared A. Bose· 1 shared A. Ghosh· 1 shared A. K. Patel· 1 shared A. K. Shaw· 1 shared A. Maitra· 1 shared A. Mondal· 1 shared B. Sinha· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperBiophysics

A. Behera

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

TGFb-ANGPT2-Tie2 axis in cancer-associated fibroblasts reprograms oral cancer cells to embryonic-like cell state with predictive significance of poor prognosis

P. Mitra, U. Saha, K. J. Stephen, P. Prasad, A. K. Patel, B. V. Harshvardhan, S. K. Mondal, S. Kurkalang, S. Roy, A. Ghosh, S. S. Roy, J. DasSarma, N. K. Biswas, M. Acharya, R. Sharan, , S. Singh

The paper identifies the TGFβ-ANGPT2-Tie2 axis in cancer-associated fibroblasts that reprograms oral cancer cells to an embryonic-like state, linked to poor prognosis.

bioRxiv · Jun 29, 2024 · PreprintCancer Biology

Connexin 43 mediated collective cell migration is independent of Golgi orientation

M. Sharma, S. Mukherjee, A. K. Shaw, A. Mondal, A. Behera, J. Das, A. Bose, B. Sinha, J. DasSarma

This study shows that Connexin 43 promotes collective cell migration by reducing Golgi reorientation, independent of directed secretory trafficking.

bioRxiv · May 3, 2023Biophysics

Matrix metalloproteinases and tissue inhibitors of metalloproteinases in murine coronavirus-induced neuroinflammation

S. Sengupta, S. Addya, D. Biswas, J. DasSarma

This paper investigates the role of MMPs and TIMP-1 in MHV-induced neuroinflammation, showing their involvement in inflammation and potential as therapeutic targets.

bioRxiv · Sep 17, 2020Microbiology