Author

J. Ermann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Assay:flow cytometry· 2 Organism:mouse· 2 Assay:clinical cohort / trial· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

A. Alhasan· 1 shared A. R. Patel· 1 shared C. Shukla· 1 shared D. Wu· 1 shared E. Haley· 1 shared E. Weldon· 1 shared H. Alfassam· 1 shared I. N. Hossain· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperBioengineering

A. Alhasan

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A Mechanically Resilient Soft Hydrogel Improves Drug Delivery for Treating Post-Traumatic Osteoarthritis in Physically Active Joints

N. Joshi, J. Yan, M. Dang, K. Slaughter, Y. Wang, D. Wu, T. Ung, V. Pandya, M. X. Chen, S. Kaur, S. Bhagchandani, H. Alfassam, J. Joseph, J. Gao, M. Dewani, , J. Ermann

This paper develops a mechanically resilient hydrogel for drug delivery in physically active joints, showing sustained release and reduced cartilage degeneration in a mouse model of post-traumatic osteoarthritis.

bioRxiv · May 16, 2024Bioengineering

Polyfunctional IL-17A+ MAIT cells are expanded in the peripheral blood of patients with HLA-B27+ axial spondyloarthritis

M. Lefton, N. Sharma, A. R. Patel, J. A. Sparks, J. Ermann

This study identifies an expansion of polyfunctional IL-17A+ MAIT cells in HLA-B27+ axSpA patients, linking intestinal inflammation to axial spondyloarthritis pathogenesis.

bioRxiv · Jan 17, 2022 · PreprintImmunology

The impact of genetic background and sex on the phenotype of IL-23 induced murine arthritis

E. Haley, M. Matmusaev, I. N. Hossain, S. Davin, T. M. Martin, J. Ermann

This study investigates how genetic background and sex affect IL-23-induced arthritis in mice, finding sex differences and genetic strain-specific disease manifestations.

bioRxiv · Feb 3, 2021Immunology