Author

J. Panov

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Assay:metabolomics· 2 Assay:proteomics· 2

Frequent Co-Authors

M. Levi· 3 shared A. Z. Rosenberg· 2 shared B. A. Jones· 2 shared J. B. Kopp· 2 shared K. Myakala· 2 shared L. Brodsky· 2 shared U. Guha· 2 shared A. Cheema· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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3 shared papersPathology

M. Levi

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Author profile →

3 papers

Recent·Most cited

Estrogen-related receptor agonism reverses mitochondrial dysfunction and inflammation in the aging kidney

X. X. Wang, K. Myakala, A. E. Libby, S. Ranjit, S. Takahashi, B. A. Jones, K. Bhasin, Y. Qi, K. Krausz, P. Zerfas, J. Panov, t. J. velenosi, d. P. patel, P. Daneshpajouhnejad, A. Titievsky, , M. Levi

This study shows that ERR agonism improves mitochondrial function and reduces inflammation in aging kidneys, while STING inhibition also reduces inflammation and increases mitochondrial gene expression.

The American Journal of Pathology · Dec 1, 2023Peer-reviewedPathology

NAD metabolism modulates inflammation and mitochondria function in diabetic kidney disease

K. Myakala, X. Wang, N. V. Shults, B. A. Jones, X. Yang, A. Z. Rosenberg, B. Ginley, P. Sarder, L. Brodsky, Y. Jang, C. H. Na, Y. Chi, X. Zhang, U. Guha, C. Wu, , M. Levi

This study shows that nicotinamide riboside (NR) improves mitochondrial function and reduces inflammation in diabetic kidney disease by enhancing NAD metabolism and inhibiting the cGAS-STING pathway.

bioRxiv · Dec 5, 2021Pathology

Adenovirus transduction to express human ACE2 causes obesity-specific morbidity in mice, impeding studies on the effect of host nutritional status on SARS-CoV-2 pathogenesis.

P. Rai, C. Chuong, T. LeRoith, J. W. Smyth, N. K. Duggal, J. Weger-Lucarelli, J. Panov, M. Levi, K. K. Hall

This study shows that adenovirus-mediated ACE2 expression in mice causes obesity-specific morbidity, complicating research on SARS-CoV-2 pathogenesis in different nutritional states.

bioRxiv · May 26, 2021Pathology