Author

J. Ryu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Microbiology· 2 Immunology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:host-pathogen & immune response· 4 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Assay:proteomics· 2 Organism:bacteria (other)· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

J. W. Jung· 2 shared S. Kim· 2 shared A. Abe· 1 shared A. Hasan· 1 shared C. Sasakawa· 1 shared D.-G. Song· 1 shared E. M. Kim· 1 shared E.-A. Shin· 1 shared

Activity by Year

2019-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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2 shared papersPhysiology

J. W. Jung

Mutual co-authors in cluster

Author profile →

6 papers

Recent·Most cited

Acceleration of HDL-mediated cellular cholesterol efflux alleviates periodontitis

J.-h. Ryu, T.-T. Tran, G. Lee, Y. H. Huh, K.-H. Chung, S. Y. Lee, K. H. Park, M.-S. Kook, J. Ryu, O.-S. Kim, H.-P. Lim, J.-T. Koh

This study shows hypercholesterolemia contributes to periodontitis and that fenofibrate reduces disease severity by enhancing HDL-mediated cholesterol efflux.

bioRxiv · Jan 18, 2024 · PreprintCell Biology

Intestinal epithelial IPMK protects mice from experimental colitis via governing colonic tuft cell development

S. E. Park, J. W. Jung, S.-H. Lee, S. J. Park, J. Ryu, S. K. Oh, S. Fang, S. Kim

This study shows that IPMK in intestinal epithelial cells protects against colitis by regulating tuft cell development and cholinergic signaling.

bioRxiv · Sep 15, 2021 · PreprintPhysiology

CBL-C E3 ubiquitin ligase acts as a host defense to mediate ubiquitin-proteasomal degradation of enteropathogenic Escherichia coli Tir protein

J. Ryu, R. Otsubo, H. Ashida, T. Iida, A. Abe, C. Sasakawa, H. Mimuro

The paper shows that CBL-C ubiquitin ligase targets Tir protein for proteasomal degradation, reducing EPEC colonization.

bioRxiv · Mar 8, 2021 · PreprintMicrobiology

Differential TM4SF5-mediated SIRT1 modulation and signaling for chronic liver disease

J. Ryu, E. M. Kim, M. Kang, D.-G. Song, E.-A. Shin, J. W. Jung, S.-H. Nam, J. E. Kim, H.-J. Kim, J.-H. Lee, J.-H. Yoon, T. Son, S. Kim, H. Y. Kim, J. W. Lee

This study identifies TM4SF5's role in modulating SIRT1 and STAT3 signaling during NAFL to NASH progression, showing age-dependent effects and potential therapeutic targets.

bioRxiv · Feb 19, 2020 · PreprintCell Biology

(p)ppGpp directly regulates translation initiation during entry into quiescence

S. Diez, J. Ryu, K. Caban, R. L. Gonzalez, J. Dworkin

This study shows that (p)ppGpp inhibits protein synthesis in Bacillus subtilis during quiescence by targeting the GTPase IF2, preventing its allosteric activation during translation initiation.

bioRxiv · Oct 17, 2019Microbiology

An insect serotonin receptor mediates cellular immune responses and its inhibition by phenylethylamide derivatives from bacterial secondary metabolites

Y. Kim, A. Hasan, H.-S. Yeom, J. Ryu, H. Bode

This study identifies a serotonin receptor in beet armyworms and shows bacterial metabolites inhibit it, leading to immunosuppression.

bioRxiv · Feb 20, 2019 · PreprintImmunology