Author

S. L. Teitelbaum

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pathology· 1 Pharmacology & Toxicology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Y. Tanaka· 2 shared A. K. Oestreich· 1 shared A. R. Osterburg· 1 shared B. S. Coller· 1 shared C. A. Harris· 1 shared C. Locuson· 1 shared C. M. Shaver· 1 shared C. T. N. Pham· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersPathology

Y. Tanaka

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Pulmonary osteoclast-like cells in silica induced pulmonary fibrosis

Y. Hasegawa, J. M. Franks, Y. Tanaka, Y. Uehara, D. F. Read, C. Williams, S. Srivatsan, L. B. Pitstick, N. M. Nikolaidis, C. M. Shaver, H. Wu, J. C. Gardner, A. R. Osterburg, J. J. Yu, E. J. Kopras, , F. X. McCormack

The paper investigates silica-induced pulmonary fibrosis in mice, finding that silica triggers osteoclast-like differentiation of macrophages and monocytes, contributing to lung injury and fibrosis.

bioRxiv · Feb 17, 2023Pathology

Adipose Tissue is a Critical Regulator of Osteoarthritis

K. H. Collins, K. L. Lenz, E. N. Pollitt, D. Ferguson, I. Hutson, L. E. Springer, A. K. Oestreich, R. Tang, Y.-R. Choi, G. A. Meyer, S. L. Teitelbaum, C. T. N. Pham, C. A. Harris, F. Guilak

This study shows adipose tissue, not body weight, drives osteoarthritis progression through paracrine signaling in a mouse model of lipodystrophy.

bioRxiv · Jun 4, 2020Physiology

Novel Pure αVβ3 Integrin Antagonists That Do Not Induce Receptor Extension, Prime the Receptor, or Enhance Angiogenesis at Low Concentrations

J. Li, Y. Fukase, Y. Shang, W. Zou, J. Munoz-Felix, L. Buitrago, J. van Agthoven, Y. Zhang, R. Hara, Y. Tanaka, R. Okamoto, T. Yasui, T. Nakahata, T. Imaeda, K. Aso, , B. S. Coller

This paper designs and evaluates two small-molecule Vβ3 antagonists that inhibit cell adhesion and bone resorption without inducing receptor conformational changes or enhancing angiogenesis at low concentrations.

bioRxiv · May 24, 2019Pharmacology & Toxicology