Author

B. Ning

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cell Biology· 2 Bioengineering· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

A. Zhang· 1 shared C. Cai· 1 shared C. Laporte· 1 shared C. Zhou· 1 shared H. Li· 1 shared H. Shengjie· 1 shared I. Londono· 1 shared I. Villemure· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

A. Zhang

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Zoledronate reduces loading-induced microdamage in cortical ulna of ovariectomized rats

B. Ning, I. Londono, C. Laporte, I. Villemure

This study shows zoledronate reduces microdamage accumulation in cortical bone of ovariectomized rats under loading, improving bone structural properties and formation.

bioRxiv · May 12, 2023Bioengineering

DEPDC1/ EEF1A1 complex promotes the progression of human osteosarcoma via downregulation of FOXO3a

L. Shen, H. Li, A. Zhang, R. Liu, C. Zhou, Y. Zhang, K. Zhao, M. Bretches, L. Lu, S.-Y. Yang, B. Ning

This study identifies the DEPDC1-EEF1A1-FOXO3a axis as a critical pathway promoting osteosarcoma progression by downregulating FOXO3a, with implications for therapeutic strategies.

bioRxiv · Apr 14, 2021 · PreprintCell Biology

Liver X Receptor; Controls Hepatic Stellate Cell Activation via Hedgehog Signaling

L. Zhong, B. Ning, X. Du, H. Shengjie, C. Cai, Y. Zhou, L. Deng, W. Shen

This study shows that LXRβ suppresses hepatic stellate cell activation via inhibition of Hedgehog signaling, offering a potential therapeutic target for liver fibrosis without causing liver steatosis.

bioRxiv · Jan 8, 2021 · PreprintCell Biology