Author

B. S. Katzenellenbogen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Biophysics· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:drug response / drug discovery· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:genetic perturbation / CRISPR· 1 Approach:mechanistic / dynamical model· 1 Organism:mouse· 1 Research Focus:protein folding & dynamics· 1

Frequent Co-Authors

J. A. Katzenellenbogen· 2 shared S. H. Kim· 2 shared A. N. Mogol· 1 shared A. Papa· 1 shared A. S. Casiano· 1 shared A. S. Raghavendra· 1 shared B. H. Park· 1 shared C. A. Natale· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersBiophysics

J. A. Katzenellenbogen

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Asymmetric Allostery in Estrogen Receptor-α Homodimers Drives Responses to the Ensemble of Estrogens in the Hormonal Milieu

C. K. Min, J. C. Nwachukwu, Y. Hou, R. Russo, A. Papa, J. Min, R. Peng, S. H. Kim, Y. Zeigler, E. S. Rangarajan, T. Izard, B. S. Katzenellenbogen, J. A. Katzenellenbogen, K. W. Nettles

This paper shows that estrogen receptor-α homodimers can function as conformational heterodimers, integrating different ligand signals to produce distinct agonist or antagonist activity profiles.

bioRxiv · Apr 10, 2024 · PreprintBiophysics

Targeting metabolic adaptations in the breast cancer liver metastatic niche using dietary approaches to improve endocrine therapy efficacy

Q. Zuo, A. N. Mogol, Y.-J. Liu, A. S. Casiano, C. Chien, J. Drnevich, O. B. Imir, E. Kulkoyluoglu-Cotul, N. H. Park, D. J. Shapiro, B. H. Park, Y. Ziegler, B. S. Katzenellenbogen, E. Aranda, J. D. O'Neill, , Z. Madak-Erdogan

This study identifies a mechanism of endocrine resistance in liver metastases of ER+ breast cancer and shows that a fasting-mimicking diet enhances the efficacy of fulvestrant treatment.

bioRxiv · Sep 7, 2021Cancer Biology

CHRM1 is a Druggable Melanoma Target Whose Endogenous Activity is Determined by Inherited Genetic Variation in DOPA Production

M. Doepner, C. A. Natale, I. Y. Lee, S. Venkat, S. H. Kim, J. A. Katzenellenbogen, B. S. Katzenellenbogen, M. Feigin, T. W. Ridky

This study identifies CHRM1 as a druggable melanoma target influenced by genetic variation in DOPA production, showing DOPA inhibits CHRM1 to suppress melanoma proliferation.

bioRxiv · Mar 3, 2021 · PreprintCancer Biology