Author

D. Bronder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:chromatin & epigenetic regulation· 2 Approach:statistical model· 2 Assay:whole-genome sequencing· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1

Frequent Co-Authors

D. Hirsch· 2 shared D. Wangsa· 2 shared K. Heselmeyer-Haddad· 2 shared T. Ried· 2 shared A. Guyer· 1 shared A. Lischka· 1 shared A. Mazzagatti· 1 shared A. Nussenzweig· 1 shared

Activity by Year

2019-2023 · peak 1/yr

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10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

D. Hirsch

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3 papers

Recent·Most cited

Mitotic clustering of pulverized chromosomes from micronuclei

Y.-F. Lin, J. E. Valle-Inclan, A. Mazzagatti, Q. Hu, E. G. Maurais, A. Guyer, J. T. Sanders, J. Engel, G. Nguyen, D. Bronder, S. F. Bakhoum, I. Cortes-Ciriano, P. Ly

This study shows that acentric chromosome fragments from micronuclei cluster during mitosis, leading to biased inheritance and balanced chromothripsis in cancer.

Nature · Jun 29, 2023Peer-reviewedCell Biology

TP53 loss initiates chromosomal instability in high-grade serous ovarian cancer

D. Bronder, D. Wangsa, D. Zong, T. J. Meyer, R. Wardenaar, P. Minshall, A. Tighe, D. Hirsch, K. Heselmeyer-Haddad, L. Nelson, D. Spierings, J. C. McGrail, M. Cam, A. Nussenzweig, F. Foijer, , S. S. Taylor

This study shows that TP53 loss in fallopian tube epithelial cells drives chromosomal instability through cell cycle deregulation, without requiring additional mutations.

bioRxiv · Mar 12, 2021Cancer Biology

Cancer-type specific aneuploidies hard-wire chromosome-wide gene expression patterns of their tissue of origin

N. Auslander, K. Heselmeyer-Haddad, S. Patkar, D. Hirsch, J. Camps, M. Brown, D. Bronder, W.-D. Chen, R. Lokanga, D. Wangsa, D. Wangsa, Y. Hu, A. Lischka, R. Braun, G. Emons, , t. ried

The paper shows cancer-type specific aneuploidies correlate with normal tissue gene expression patterns, suggesting they hard-wire chromosome-wide expression levels to support tumorigenesis.

bioRxiv · Mar 1, 2019Cancer Biology