Author

J. Magallanes

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Pathology· 1

Signature Assays, Organisms & Methods

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Research Focus:development & morphogenesis· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

D. Evseenko· 3 shared A. Sarkar· 2 shared B. Van Handel· 2 shared F. Bian· 2 shared J. Lu· 2 shared J. Tassey· 2 shared J. Zhang· 2 shared N. Q. Liu· 2 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCell Biology

D. Evseenko

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Inactivation of a non-canonical gp130 signaling arm attenuates chronic systemic inflammation and multimorbidity induced by a high-fat diet.

Y. Lee, A. Sarkar, J. Tassey, J. N. Levi, S. Lee, N. Q. Liu, A. C. Drake, J. Magallanes, J. Lu, D. Ge, H. Tang, T. Mkaratigwa, F. Bian, R. Shkhyan, M. A. Bonaguidi, D. Evseenko

This study shows that inhibiting a non-canonical gp130 signaling pathway reduces chronic inflammation and multimorbidity in a high-fat diet model.

bioRxiv · Apr 8, 2024 · PreprintCell Biology

Gli1+ mesenchymal progenitors contribute to multilineage differentiation in a mouse model of post-traumatic joint injury

J. Magallanes, N. Q. Liu, J. Zhang, Y. Ouyang, T. Mkaratigwa, F. Bian, B. Van Handel, T. Skorka, F. A. Petrigliano, D. Evseenko

This study establishes a mouse model of post-traumatic joint injury and identifies Gli1+ mesenchymal progenitors as key contributors to arthrofibrosis and heterotopic ossification.

bioRxiv · Jun 11, 2022 · PreprintPathology

gp130/STAT3 signaling is required for homeostatic proliferation and anabolism in postnatal growth plate and articular chondrocytes

N. Liu, Y. Lin, L. Li, J. Lu, D. Geng, J. Zhang, T. Jashashvili, Z. Buser, J. Magallanes, J. Tassey, R. Shkhyan, A. Sarkar, N. Lopez, L. Wang, F. Petrigliano, , D. Evseenko

This study shows that gp130/STAT3 signaling is essential for chondrocyte proliferation and anabolism, with loss leading to growth defects and cartilage dysfunction.

bioRxiv · Oct 12, 2021Cell Biology