Author

J. Querol

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cancer & tumor microenvironment· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Assay:ChIP-seq· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:statistical model· 2

Frequent Co-Authors

J. Arribas· 3 shared L. Pascual-Reguant· 3 shared S. Peiro· 3 shared T. H. Stracker· 3 shared G. Serra-Bardenys· 2 shared G. Verde· 2 shared P. Nuciforo· 2 shared R. Aiese Cigliano· 2 shared

Activity by Year

2018-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersCancer Biology

J. Arribas

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

GEMC1 and MCIDAS interactions with SWI/SNF complexes regulate the multiciliated cell-specific transcriptional program

M. Lewis, B. Terre, P. A. Knobel, T. Cheng, H. Lu, C. S. Attolini, J. Smak, E. Coyaud, I. Garcia-Cao, J. Querol, G. Gil-Gomez, G. Piergiovanni, V. Costanzo, S. Peiro, B. Raught, , T. H. Stracker

This study identifies distinct interactions of GEMC1 and MCIDAS with SWI/SNF subcomplexes in multiciliated cell differentiation and shows that these interactions are required for MCC-specific transcriptional regulation.

bioRxiv · Dec 2, 2022 · PreprintCell Biology

The BRD4S-LOXL2-MED1 interaction at the forefront of cell cycle transcriptional control in triple-negative breast cancer

L. Pascual-Reguant, T. V. Tian, D. Datta, D. Cianferoni, S. Kourtis, A. Ganez Zapater, C. Cannata, Q. Serra Camprubi, L. Espinar, M. Guirola, J. Querol, A. Miro Canturri, J. Arribas, L. Serrano, S. Peiro, S. Sdelci

This study shows that inhibiting LOXL2 and BRD4 together reduces triple-negative breast cancer proliferation by disrupting the BRD4S-LOXL2-MED1 interaction that controls cell cycle gene expression.

bioRxiv · May 27, 2022 · PreprintCancer Biology

Maintaining oxidized H3 in heterochromatin is required for the oncogenic capacity of triple-negative breast cancer cells

G. Serra-Bardenys, T. V. Tian, E. Blanco, J. Querol, L. Pascual-Reguant, B. Morancho, M. Escorihuela, S. Segura-Bayona, G. Verde, R. Aiese Cigliano, A. Millanes-Romero, C. Jerónimo, P. Nuciforo, S. Simonetti, C. Viaplana, , S. Peiró

This study identifies the role of H3K4ox in maintaining heterochromatin structure in TNBC cells and shows its necessity for oncogenic properties.

bioRxiv · Jan 30, 2020 · PreprintCancer Biology

Reduction of LOXL2-mediated H3K4 oxidation increases chromatin accessibility and promotes chemosensitivity of triple-negative breast cancer cells

J. P. Cebria-Costa, L. Pascual-Reguant, A. Gonzalez Perez, G. Serra-Bardenys, J. Querol, M. Cosin, G. Verde, R. Aiese Cigliano, W. Sanseverino, S. Segura-Bayona, A. Iturbide, D. Andreu, P. Nuciforo, C. Bernado Morales, V. Rodilla, , S. Peiro

This study shows that reducing LOXL2-mediated H3K4 oxidation decreases chromatin compaction and increases chemosensitivity in triple-negative breast cancer cells.

bioRxiv · Sep 13, 2018 · PreprintCancer Biology