Author

S. Peiro

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1

Frequent Co-Authors

J. Querol· 2 shared T. V. Tian· 2 shared A. Ganez Zapater· 1 shared A. Klonizakis· 1 shared A. Leutz· 1 shared A. Miro Canturri· 1 shared B. Raught· 1 shared B. Terre· 1 shared

Activity by Year

2022-2022 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

J. Querol

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

GEMC1 and MCIDAS interactions with SWI/SNF complexes regulate the multiciliated cell-specific transcriptional program

M. Lewis, B. Terre, P. A. Knobel, T. Cheng, H. Lu, C. S. Attolini, J. Smak, E. Coyaud, I. Garcia-Cao, J. Querol, G. Gil-Gomez, G. Piergiovanni, V. Costanzo, S. Peiro, B. Raught, , T. H. Stracker

This study identifies distinct interactions of GEMC1 and MCIDAS with SWI/SNF subcomplexes in multiciliated cell differentiation and shows that these interactions are required for MCC-specific transcriptional regulation.

bioRxiv · Dec 2, 2022 · PreprintCell Biology

Carm1 regulates the speed of C/EBPa-induced transdifferentiation by a cofactor stealing mechanism

G. T. Garcia, E. Kowenz-Leutz, T. V. Tian, A. Klonizakis, J. Lerner, L. De Andres-Aguayo, C. Berenguer, M. P. Carmona, M. V. Casadesus, R. Bulteau, M. Francesconi, S. Peiro, K. S. Zaret, A. Leutz, T. Graf

This study shows that a C/EBP mutation accelerates B cell to macrophage transdifferentiation by altering PU.1 interactions, and that Carm1 methylation regulates this process.

bioRxiv · Oct 3, 2022Cell Biology

The BRD4S-LOXL2-MED1 interaction at the forefront of cell cycle transcriptional control in triple-negative breast cancer

L. Pascual-Reguant, T. V. Tian, D. Datta, D. Cianferoni, S. Kourtis, A. Ganez Zapater, C. Cannata, Q. Serra Camprubi, L. Espinar, M. Guirola, J. Querol, A. Miro Canturri, J. Arribas, L. Serrano, S. Peiro, S. Sdelci

This study shows that inhibiting LOXL2 and BRD4 together reduces triple-negative breast cancer proliferation by disrupting the BRD4S-LOXL2-MED1 interaction that controls cell cycle gene expression.

bioRxiv · May 27, 2022 · PreprintCancer Biology