Author

H. Weng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

C. Shao· 2 shared F. Link· 2 shared H. Ding· 2 shared H. Liu· 2 shared M. Ebert· 2 shared S. Dooley· 2 shared S. Munker· 2 shared S. Wang· 2 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

C. Shao

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Retinoic acid generates a beneficial microenvironment for liver progenitor cell activation in acute liver failure

S. Wang, F. Link, S. Munker, W. Wang, R. Feng, R. Lieber, Y. Li, Y. Yao, H. Liu, C. Shao, M. Ebert, H. Ding, S. Dooley, H. Weng, S. Wang

This study shows retinoic acid secreted by activated hepatic stellate cells promotes liver progenitor cell activation in acute liver failure, improving patient outcomes.

bioRxiv · Jan 23, 2024Cell Biology

ECM1 Attenuates Hepatic Fibrosis by Interfering with Mediators of Latent TGF-β1 Activation

F. Link, Y. Li, J. Zhao, S. Munker, W. Fan, Z. Nwosu, Y. Yao, S. Wang, C. Huang, S. Hammad, R. Liebe, H. Liu, C. Shao, C. Gao, B. Sun, , S. Wang

This study shows ECM1 prevents hepatic fibrosis by inhibiting TGF-β1 activation, with implications for developing safe anti-fibrotic therapies.

bioRxiv · Dec 12, 2023Cell Biology

WDR76 promotes MLL-rearranged leukemia via selective recognition of 5-hydroxymethylcytosine in DNA

K. E. Malecek, H. Weng, M. A. Sullivan, C. Y. Kokontis, M. S. Werner, J. Chen, A. J. Ruthenburg

This study identifies WDR76 as a hmC-specific protein that promotes MLL-rearranged leukemia by modulating gene expression and facilitating leukemogenesis.

bioRxiv · Jan 3, 2019 · PreprintBiochemistry