Author

J. Blenis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 3 Organism:human· 3 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:metabolism & bioenergetics· 1 Assay:metabolomics· 1

Frequent Co-Authors

A. Ahmed· 1 shared A. Montalbano· 1 shared A. Mutvei· 1 shared A. P. Gomes· 1 shared A. Rosenzweig· 1 shared A. Unni· 1 shared B. E. Schaffer· 1 shared C. Kinzig· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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1 shared paperCancer Biology

A. Ahmed

Mutual co-authors in cluster

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3 papers

Recent·Most cited

LKB1 suppresses growth and promotes the internalization of EGFR through the PIKFYVE lipid kinase

J. Ferrarone, J. Thomas, A. Unni, Y. Zheng, M. Nagiec, E. Gardner, O. Mashadova, K. Li, N. Koundouros, A. Montalbano, M. Mustafa, L. Cantley, J. Blenis, N. Sanjana, H. Varmus

This study identifies LKB1's role in suppressing growth by activating PIKFYVE to promote EGFR internalization in LUAD.

bioRxiv · Oct 19, 2023 · PreprintCancer Biology

Glutamine deprivation triggers NAGK-dependent hexosamine salvage

S. L. Campbell, C. Mesaros, L. Izzo, H. Affronti, T. Tsang, M. Noji, B. E. Schaffer, K. Sun, S. Trefely, S. Kruijning, J. Blenis, I. A. Blair, K. E. Wellen

This study shows that glutamine deprivation in pancreatic ductal adenocarcinoma cells increases free GlcNAc and activates NAGK-dependent hexosamine salvage to support tumor growth.

bioRxiv · Sep 13, 2020Cancer Biology

Age-induced methylmalonic acid accumulation promotes tumor progression and aggressiveness

A. P. Gomes, D. Ilter, V. Low, J. Endress, J. Fernandez-Garcia, A. Rosenzweig, T. Schild, D. Broekaert, A. Ahmed, M. Planque, I. Elia, J. Han, C. Kinzig, E. Mullarky, A. Mutvei, , J. Blenis

The paper shows that methylmalonic acid accumulation with age promotes tumor progression by inducing SOX4 and reprogramming cancer cells.

bioRxiv · Jun 4, 2020 · PreprintCancer Biology