Author

K. Inoki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 3 Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:development & morphogenesis· 1 Assay:imaging· 1

Frequent Co-Authors

A. Benchamana· 1 shared A. Das· 1 shared A. Inoki· 1 shared A. Nishii· 1 shared B. Learman· 1 shared C. Corsa· 1 shared C. Dugan· 1 shared C. Evans· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperPhysiology

A. Benchamana

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Identification of Circular Dorsal Ruffles as Signal Platforms for the AKT pathways in Glomerular Podocytes

S. Yoshida, R. Hua, M. Torres, X. Sun, J. Wei, L. Wang, K. Inoki

The paper identifies circular dorsal ruffles as signal platforms for the AKT-mTORC1 pathway in glomerular podocytes, linking them to kidney function and potential therapeutic targets.

Journal of Cellular Physiology · Mar 16, 2023Peer-reviewedCell Biology

The molecular and metabolic program for adaptation of white adipocytesto cool physiologic temperatures

O. A. MacDougald, H. Mori, C. Dugan, A. Nishii, A. Benchamana, Z. Li, T. Cadenhead, A. Das, C. Evans, K. Overmyer, S. Romanelli, S. Peterson, D. Bagchi, C. Corsa, J. Hardij, , K. Inoki

This study investigates how white adipocytes adapt to cooler temperatures, finding changes in gene expression, lipid metabolism, and energy source utilization.

bioRxiv · Oct 16, 2020 · PreprintPhysiology

Endoplasmic reticulum associated degradation preserves hematopoietic stem cell quiescence and self-renewal by restricting mTOR activity

L. Liu, A. Inoki, K. Fan, F. Mao, G. Shi, X. Jin, M. Zhao, G. Ney, S. Sun, Y. Dou, K. Inoki, L. Qi, Q. Li

This study shows that ERAD preserves HSC quiescence and self-renewal by restricting mTOR activity, preventing HSC depletion and hyper-proliferation.

bioRxiv · Jul 21, 2019Cell Biology