Author

A. Parks

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Organism:bacteria (other)· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

G. S. Heo· 2 shared Y. Liu· 2 shared A. Bhambhani· 1 shared A. Bredemeyer· 1 shared A. E. Gelman· 1 shared A. Marconi· 1 shared A. Mercurio· 1 shared A. S. Krupnick· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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2 shared papersImmunology

G. S. Heo

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Expansion of Disease Specific Cardiac Macrophages in Immune Checkpoint Inhibitor Myocarditis

P. Ma, J. Liu, J. Qin, L. Lai, G. S. Heo, H. Luehmann, D. Sultan, A. Bredemeyer, G. Bajpai, G. Feng, J. Jimenez, A. Parks, J. Amrute, A. Villanueva, Y. Liu, , K. Lavine

This study identifies an inflammatory macrophage population expanded in ICI myocarditis, driven by IFN-γ signaling and T-cell interactions, suggesting IFN-γ blockade as a potential therapeutic strategy.

bioRxiv · Apr 28, 2023 · PreprintImmunology

Reprogramming Alveolar Macrophage Responses to TGF-β Reveals CCR2+ Monocyte Activity that Promotes Bronchiolitis Obliterans Syndrome

Z. Liu, F. Liao, J. Zhu, D. Zhou, G. S. Heo, H. P. Luehmann, D. Scozzi, A. Parks, R. Hachem, D. Byers, L. K. Tague, H. S. Kulkarni, M. Cano, B. W. Wong, W. Li, , A. E. Gelman

This study identifies alveolar macrophage reprogramming by ECP that reduces TGF-β bioavailability and prevents BOS by inhibiting CCR2+ monocyte-driven inflammation.

bioRxiv · Jan 27, 2022 · PreprintImmunology

Bacteriophage SP01 Gene Product 56 (gp56) Inhibits Bacillus subtilis Cell Division by Interacting with DivIC/FtsL to Prevent Pbp2B/FtsW Recruitment

A. Bhambhani, I. Iadicicco, J. Lee, S. Ahmed, M. Belfatto, D. Held, A. Marconi, A. Parks, C. Stewart, W. Margolin, P. A. Levin, D. P. Haeusser

This paper identifies gp56, a bacteriophage protein, as an inhibitor of Bacillus subtilis cell division by blocking recruitment of late division proteins through interaction with DivIC/FtsL.

Journal of Bacteriology · Dec 18, 2020Peer-reviewedMicrobiology

Tumor cell-organized fibronectin is required to maintain a dormant breast cancer population

L. Barney, C. Hall, A. Schwartz, A. Parks, C. Sparages, S. Galarza, M. Platt, A. Mercurio, S. Peyton

This study identifies tumor cell-organized fibronectin as essential for maintaining dormant breast cancer populations through in vitro experiments.

bioRxiv · Jul 1, 2019 · PreprintCancer Biology