Author

M. Tracy

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. Bode· 1 shared A. Holder· 1 shared A. J. Herr· 1 shared A. M. Loeb· 1 shared A. R. Chia· 1 shared B. Hearn· 1 shared C. Weber· 1 shared F. Khalili-Araghi· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperCancer Biology

A. Bode

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Inflammation-Induced Claudin-2 Upregulation Limits Pancreatitis Progression by Enhancing Tight Junction-Controlled Pancreatic Ductal Transport

S. Kesaraju, Y. Li, J. Xing, M. Tracy, K. Wannemo, A. Holder, P. Zhao, M. A. Khan, J. Kainov, N. Rana, M. Sidahmed, S. Hyoju, L. Smith, J. Matthews, S. Tay, , C. Weber

This study shows CLDN2 upregulation during pancreatitis limits disease progression by enhancing tight junction-controlled fluid transport in pancreatic ductal epithelium.

bioRxiv · Sep 1, 2023 · PreprintPathology

mt-LAF3 is a pseudouridine synthase ortholog required for mitochondrial rRNA and mRNA gene expression in Trypanosoma brucei

S. McDermott, V. Pham, I. Lewis, M. Tracy, K. Stuart

This study shows that mt-LAF3 is essential for mitochondrial rRNA and mRNA expression in T. brucei but lacks PUS catalytic activity, instead functioning as an RNA-stabilizing scaffold.

bioRxiv · Feb 23, 2023 · PreprintMicrobiology

Spontaneous polyploids and antimutators compete during the evolution of mutator cells

M. Tracy, M. Lee, B. Hearn, I. Dowsett, L. Thurber, J. Loo, A. M. Loeb, K. Preston, M. Tuncel, N. Ghodsian, A. Bode, T. Tang, A. R. Chia, A. J. Herr

The paper shows that spontaneous polyploids and antimutator clones outcompete each other in mutator yeast cultures, with polyploids dominating in haploid and antimutators in diploid settings.

bioRxiv · May 9, 2020Cancer Biology